Rui T - Search Results

1

miR‑141 impairs mitochondrial function in cardiomyocytes subjected to hypoxia/reoxygenation by targeting Sirt1 and MFN2.

Zhang H, Wang Y, Wu K, Liu R, Wang H, Yao Y, Kvietys P, Rui T. Zhang H, et al. Among authors: rui t. Exp Ther Med. 2022 Nov 14;24(6):763. doi: 10.3892/etm.2022.11699. eCollection 2022 Dec. Exp Ther Med. 2022. PMID: 36561976 Free PMC article.

2

NFkappaB and AP-1 differentially contribute to the induction of Mn-SOD and eNOS during the development of oxidant tolerance.

Rui T, Kvietys PR. Rui T, et al. FASEB J. 2005 Nov;19(13):1908-10. doi: 10.1096/fj.05-4028fje. Epub 2005 Sep 12. FASEB J. 2005. PMID: 16157693

3

Experimental diabetes mellitus exacerbates ischemia/reperfusion-induced myocardial injury by promoting mitochondrial fission: Role of down-regulation of myocardial Sirt1 and subsequent Akt/Drp1 interaction.

Tao A, Xu X, Kvietys P, Kao R, Martin C, Rui T. Tao A, et al. Among authors: rui t. Int J Biochem Cell Biol. 2018 Dec;105:94-103. doi: 10.1016/j.biocel.2018.10.011. Epub 2018 Oct 28. Int J Biochem Cell Biol. 2018. PMID: 30381241

4

[No title available]

[No authors listed] [No authors listed] PMID: 35553777

5

NADPH oxidase contributes to conversion of cardiac myocytes to a proinflammatory phenotype in sepsis.

Wu J, Xu H, Yang M, Martin CM, Kvietys PR, Rui T. Wu J, et al. Among authors: rui t. Free Radic Biol Med. 2009 May 15;46(10):1338-45. doi: 10.1016/j.freeradbiomed.2009.02.012. Epub 2009 Feb 26. Free Radic Biol Med. 2009. PMID: 19249346

6

Cardiomyocyte-fibroblast interaction contributes to diabetic cardiomyopathy in mice: Role of HMGB1/TLR4/IL-33 axis.

Tao A, Song J, Lan T, Xu X, Kvietys P, Kao R, Martin C, Rui T. Tao A, et al. Among authors: rui t. Biochim Biophys Acta. 2015 Oct;1852(10 Pt A):2075-85. doi: 10.1016/j.bbadis.2015.07.015. Epub 2015 Jul 22. Biochim Biophys Acta. 2015. PMID: 26209013 Free article.

7

Peroxynitrite/PKR Axis Modulates the NLRP3 Inflammasome of Cardiac Fibroblasts.

Lan T, Tao A, Xu X, Kvietys P, Rui T. Lan T, et al. Among authors: rui t. Front Immunol. 2020 Sep 25;11:558712. doi: 10.3389/fimmu.2020.558712. eCollection 2020. Front Immunol. 2020. PMID: 33101273 Free PMC article.

8

Important role of p38 MAP kinase/NF-kappaB signaling pathway in the sepsis-induced conversion of cardiac myocytes to a proinflammatory phenotype.

Yang M, Wu J, Martin CM, Kvietys PR, Rui T. Yang M, et al. Among authors: rui t. Am J Physiol Heart Circ Physiol. 2008 Feb;294(2):H994-1001. doi: 10.1152/ajpheart.01044.2007. Epub 2007 Dec 14. Am J Physiol Heart Circ Physiol. 2008. PMID: 18083899 Free article.

9

The alarmin cytokine, high mobility group box 1, is produced by viable cardiomyocytes and mediates the lipopolysaccharide-induced myocardial dysfunction via a TLR4/phosphatidylinositol 3-kinase gamma pathway.

Xu H, Su Z, Wu J, Yang M, Penninger JM, Martin CM, Kvietys PR, Rui T. Xu H, et al. Among authors: rui t. J Immunol. 2010 Feb 1;184(3):1492-8. doi: 10.4049/jimmunol.0902660. Epub 2009 Dec 18. J Immunol. 2010. PMID: 20028656

10

Cardiac fibroblasts contribute to myocardial dysfunction in mice with sepsis: the role of NLRP3 inflammasome activation.

Zhang W, Xu X, Kao R, Mele T, Kvietys P, Martin CM, Rui T. Zhang W, et al. Among authors: rui t. PLoS One. 2014 Sep 12;9(9):e107639. doi: 10.1371/journal.pone.0107639. eCollection 2014. PLoS One. 2014. PMID: 25216263 Free PMC article.

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