De Pont JJ - Search Results

1

Receptor-evoked Ca2+ mobilization in pancreatic acinar cells: evidence for a regulatory role of protein kinase C by a mechanism involving the transition of high-affinity receptors to a low-affinity state.

Willems PH, Van Hoof HJ, Van Mackelenbergh MG, Hoenderop JG, Van Emst-De Vries SE, De Pont JJ. Willems PH, et al. Among authors: de pont jj. Pflugers Arch. 1993 Jul;424(2):171-82. doi: 10.1007/BF00374609. Pflugers Arch. 1993. PMID: 7692387

2

Ca2(+)-sensitivity of inositol 1,4,5-trisphosphate-mediated Ca2+ release in permeabilized pancreatic acinar cells.

Willems PH, De Jong MD, De Pont JJ, Van Os CH. Willems PH, et al. Among authors: de pont jj, de jong md. Biochem J. 1990 Feb 1;265(3):681-7. doi: 10.1042/bj2650681. Biochem J. 1990. PMID: 2306208 Free PMC article.

3

Inhibition of inositol 1,4,5-trisphosphate-induced Ca2+ release in permeabilized pancreatic acinar cells by hormonal and phorbol ester pretreatment.

Willems PH, Van den Broek BA, Van Os CH, De Pont JJ. Willems PH, et al. Among authors: de pont jj. J Biol Chem. 1989 Jun 15;264(17):9762-7. J Biol Chem. 1989. PMID: 2785996 Free article.

4

Cholecystokinin-stimulated enzyme secretion from dispersed rabbit pancreatic acinar cells: phosphorylation-dependent changes in potency and efficacy.

Willems PH, Van Emst-de Vries SE, De Pont JJ. Willems PH, et al. Among authors: de pont jj. Pflugers Arch. 1995 Sep;430(5):626-35. doi: 10.1007/BF00386156. Pflugers Arch. 1995. PMID: 7478913 Free article.

5

Heterogeneity between intracellular Ca2+ stores as the underlying principle of quantal Ca2+ release by inositol 1,4,5-trisphosphate in permeabilized pancreatic acinar cells.

van de Put FH, De Pont JJ, Willems PH. van de Put FH, et al. Among authors: de pont jj. J Biol Chem. 1994 Apr 29;269(17):12438-43. J Biol Chem. 1994. PMID: 7513688 Free article.

6

Dose-dependent recruitment of pancreatic acinar cells during receptor-mediated calcium mobilization.

Willems PH, Van Emst-De Vries SE, Van Os CH, De Pont JJ. Willems PH, et al. Among authors: de pont jj. Cell Calcium. 1993 Feb;14(2):145-59. doi: 10.1016/0143-4160(93)90084-j. Cell Calcium. 1993. PMID: 7681361

7

Ruthenium red selectively depletes inositol 1,4,5-trisphosphate-sensitive calcium stores in permeabilized rabbit pancreatic acinar cells.

van de Put FH, Hoenderop JG, De Pont JJ, Willems PH. van de Put FH, et al. Among authors: de pont jj. J Membr Biol. 1993 Aug;135(2):153-63. doi: 10.1007/BF00231441. J Membr Biol. 1993. PMID: 7692065

8

Induction of Ca2+ oscillations by selective, U73122-mediated, depletion of inositol-trisphosphate-sensitive Ca2+ stores in rabbit pancreatic acinar cells.

Willems PH, Van de Put FH, Engbersen R, Bosch RR, Van Hoof HJ, de Pont JJ. Willems PH, et al. Among authors: de pont jj. Pflugers Arch. 1994 Jun;427(3-4):233-43. doi: 10.1007/BF00374529. Pflugers Arch. 1994. PMID: 8072841

9

CCK8-evoked Ca2+ mobilization in pancreatic acinar cells. Evidence for a regulatory role of protein kinase C by phosphorylation-dependent inhibition of signaling through the high-affinity CCK receptor.

Willems PH, van Hoof HJ, van Mackelenbergh MG, Hoenderop JG, van Emst-de Vries SE, De Pont JJ. Willems PH, et al. Among authors: de pont jj. Ann N Y Acad Sci. 1994 Mar 23;713:343-5. doi: 10.1111/j.1749-6632.1994.tb44087.x. Ann N Y Acad Sci. 1994. PMID: 8185182 No abstract available.

10

Protein kinase C activation inhibits receptor-evoked inositol trisphosphate formation and induction of cytosolic calcium oscillations by decreasing the affinity-state of the cholecystokinin receptor in pancreatic acinar cells.

Willems PH, Smeets RL, Bosch RR, Garner KM, Van Mackelenbergh MG, De Pont JJ. Willems PH, et al. Among authors: de pont jj. Cell Calcium. 1995 Dec;18(6):471-83. doi: 10.1016/0143-4160(95)90010-1. Cell Calcium. 1995. PMID: 8746946

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