Nishino H - Search Results

1

Chronically administered 3-nitropropionic acid induces striatal lesions attributed to dysfunction of the blood-brain barrier.

Nishino H, Shimano Y, Kumazaki M, Sakurai T. Nishino H, et al. Neurosci Lett. 1995 Feb 17;186(2-3):161-4. doi: 10.1016/0304-3940(95)11311-j. Neurosci Lett. 1995. PMID: 7777187

2

Chronically administered 3-nitropropionic acid produces selective lesions in the striatum and reduces muscle tonus.

Shimano Y, Kumazaki M, Sakurai T, Hida H, Fujimoto I, Fukuda A, Nishino H. Shimano Y, et al. Among authors: nishino h. Obes Res. 1995 Dec;3 Suppl 5:779S-784S. doi: 10.1002/j.1550-8528.1995.tb00499.x. Obes Res. 1995. PMID: 8653562 Free article.

3

Hypothalamic neurons are resistant to the intoxication with 3-nitropropionic acid that induces lesions in the striatum and hippocampus via the damage in the blood-brain barrier.

Nishino H, Shimano Y, Kumazaki M, Sakurai T, Hida H, Fujimoto I, Fukuda A. Nishino H, et al. Neurobiology (Bp). 1995;3(3-4):257-67. Neurobiology (Bp). 1995. PMID: 8696294

4

3-Nitropropionic acid produces striatum selective lesions accompanied by iNOS expression.

Nishino H, Fujimoto I, Shimano Y, Hida H, Kumazaki M, Fukuda A. Nishino H, et al. J Chem Neuroanat. 1996 Jun;10(3-4):209-12. doi: 10.1016/0891-0618(96)00134-2. J Chem Neuroanat. 1996. PMID: 8811425

5

Tissue extract from dopamine-depleted striatum enhances differentiation of cultured striatal type-1 astrocytes.

Shimano Y, Fukuda A, Fujimoto I, Hida H, Nakajima K, Okabe A, Nishino H. Shimano Y, et al. Among authors: nishino h. Neurosci Lett. 1996 Mar 1;205(3):193-6. doi: 10.1016/0304-3940(96)12413-7. Neurosci Lett. 1996. PMID: 8852591

6

Acute 3-nitropropionic acid intoxication induces striatal astrocytic cell death and dysfunction of the blood-brain barrier: involvement of dopamine toxicity.

Nishino H, Kumazaki M, Fukuda A, Fujimoto I, Shimano Y, Hida H, Sakurai T, Deshpande SB, Shimizu H, Morikawa S, Inubushi T. Nishino H, et al. Neurosci Res. 1997 Apr;27(4):343-55. doi: 10.1016/s0168-0102(97)01170-x. Neurosci Res. 1997. PMID: 9152047

7

Estrogen protects against while testosterone exacerbates vulnerability of the lateral striatal artery to chemical hypoxia by 3-nitropropionic acid.

Nishino H, Nakajima K, Kumazaki M, Fukuda A, Muramatsu K, Deshpande SB, Inubushi T, Morikawa S, Borlongan CV, Sanberg PR. Nishino H, et al. Neurosci Res. 1998 Apr;30(4):303-12. doi: 10.1016/s0168-0102(98)00010-8. Neurosci Res. 1998. PMID: 9678634

8

Astrocytes are more vulnerable than neurons to cellular Ca2+ overload induced by a mitochondrial toxin, 3-nitropropionic acid.

Fukuda A, Deshpande SB, Shimano Y, Nishino H. Fukuda A, et al. Among authors: nishino h. Neuroscience. 1998 Nov;87(2):497-507. doi: 10.1016/s0306-4522(98)00139-0. Neuroscience. 1998. PMID: 9740408

9

The striatum is the most vulnerable region in the brain to mitochondrial energy compromise: a hypothesis to explain its specific vulnerability.

Nishino H, Hida H, Kumazaki M, Shimano Y, Nakajima K, Shimizu H, Ooiwa T, Baba H. Nishino H, et al. J Neurotrauma. 2000 Mar;17(3):251-60. doi: 10.1089/neu.2000.17.251. J Neurotrauma. 2000. PMID: 10757330

10

Altered striatal vulnerability to 3-nitropropionic acid in rats due to sex hormone levels during late phase of brain development.

Mogami M, Hayashi Y, Masuda T, Kohri K, Nishino H, Hida H. Mogami M, et al. Among authors: nishino h. Neurosci Lett. 2008 May 16;436(3):321-5. doi: 10.1016/j.neulet.2008.03.055. Epub 2008 Mar 25. Neurosci Lett. 2008. PMID: 18406525

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