Inoue M - Search Results

1

Plasma nitric oxide end products are increased in the ischemic canine heart.

Node K, Kitakaze M, Kosaka H, Komamura K, Minamino T, Tada M, Inoue M, Hori M, Kamada T. Node K, et al. Among authors: inoue m. Biochem Biophys Res Commun. 1995 Jun 15;211(2):370-4. doi: 10.1006/bbrc.1995.1823. Biochem Biophys Res Commun. 1995. PMID: 7794246

2

Infarct size-limiting effect of ischemic preconditioning is blunted by inhibition of 5'-nucleotidase activity and attenuation of adenosine release.

Kitakaze M, Hori M, Morioka T, Minamino T, Takashima S, Sato H, Shinozaki Y, Chujo M, Mori H, Inoue M, et al. Kitakaze M, et al. Among authors: inoue m. Circulation. 1994 Mar;89(3):1237-46. doi: 10.1161/01.cir.89.3.1237. Circulation. 1994. PMID: 8124812

3

Superoxide dismutase enhances both adenosine release and coronary hyperemic flow through protection of 5'-nucleotidase against its degradation during reperfusion following ischemia in dogs.

Kitakaze M, Hori M, Takashima S, Morioka T, Minamino T, Sato H, Okazaki Y, Inoue M, Kamada T. Kitakaze M, et al. Among authors: inoue m. Biorheology. 1993 Sep-Dec;30(5-6):359-70. doi: 10.3233/bir-1993-305-607. Biorheology. 1993. PMID: 8186402

4

Reperfusion after brief ischemia disrupts the microtubule network in canine hearts.

Sato H, Hori M, Kitakaze M, Iwai K, Takashima S, Kurihara H, Inoue M, Kamada T. Sato H, et al. Among authors: inoue m. Circ Res. 1993 Feb;72(2):361-75. doi: 10.1161/01.res.72.2.361. Circ Res. 1993. PMID: 8418989

5

Ischemic preconditioning increases adenosine release and 5'-nucleotidase activity during myocardial ischemia and reperfusion in dogs. Implications for myocardial salvage.

Kitakaze M, Hori M, Takashima S, Sato H, Inoue M, Kamada T. Kitakaze M, et al. Among authors: inoue m. Circulation. 1993 Jan;87(1):208-15. doi: 10.1161/01.cir.87.1.208. Circulation. 1993. PMID: 8419009

6

Superoxide dismutase restores contractile and metabolic dysfunction through augmentation of adenosine release in coronary microembolization.

Takashima S, Hori M, Kitakaze M, Sato H, Inoue M, Kamada T. Takashima S, et al. Among authors: inoue m. Circulation. 1993 Mar;87(3):982-95. doi: 10.1161/01.cir.87.3.982. Circulation. 1993. PMID: 8443917

7

Increased release of NO during ischemia reduces myocardial contractility and improves metabolic dysfunction.

Node K, Kitakaze M, Kosaka H, Komamura K, Minamino T, Inoue M, Tada M, Hori M, Kamada T. Node K, et al. Among authors: inoue m. Circulation. 1996 Jan 15;93(2):356-64. doi: 10.1161/01.cir.93.2.356. Circulation. 1996. PMID: 8548910

8

Evidence for nitric oxide generation in the cardiomyocytes: its augmentation by hypoxia.

Kitakaze M, Node K, Komamura K, Minamino T, Inoue M, Hori M, Kamada T. Kitakaze M, et al. Among authors: inoue m. J Mol Cell Cardiol. 1995 Oct;27(10):2149-54. doi: 10.1016/s0022-2828(95)91335-1. J Mol Cell Cardiol. 1995. PMID: 8576931

9

Role of nitric oxide in regulation of coronary blood flow during myocardial ischemia in dogs.

Kitakaze M, Node K, Minamino T, Kosaka H, Shinozaki Y, Mori H, Inoue M, Hori M, Kamada T. Kitakaze M, et al. Among authors: inoue m. J Am Coll Cardiol. 1996 Jun;27(7):1804-12. doi: 10.1016/0735-1097(96)00064-2. J Am Coll Cardiol. 1996. PMID: 8636571 Free article.

10

Role of activation of protein kinase C in the infarct size-limiting effect of ischemic preconditioning through activation of ecto-5'-nucleotidase.

Kitakaze M, Node K, Minamino T, Komamura K, Funaya H, Shinozaki Y, Chujo M, Mori H, Inoue M, Hori M, Kamada T. Kitakaze M, et al. Among authors: inoue m. Circulation. 1996 Feb 15;93(4):781-91. doi: 10.1161/01.cir.93.4.781. Circulation. 1996. PMID: 8641008

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