Maruta H - Search Results

1

Molecular cloning of a novel mitogen-inducible nuclear protein with a Ran GTPase-activating domain that affects cell cycle progression.

Hattori M, Tsukamoto N, Nur-e-Kamal MS, Rubinfeld B, Iwai K, Kubota H, Maruta H, Minato N. Hattori M, et al. Among authors: maruta h. Mol Cell Biol. 1995 Jan;15(1):552-60. doi: 10.1128/MCB.15.1.552. Mol Cell Biol. 1995. PMID: 7799964 Free PMC article.

2

The role of Gln61 and Glu63 of Ras GTPases in their activation by NF1 and Ras GAP.

Nur-E-Kamal MS, Maruta H. Nur-E-Kamal MS, et al. Among authors: maruta h. Mol Biol Cell. 1992 Dec;3(12):1437-42. doi: 10.1091/mbc.3.12.1437. Mol Biol Cell. 1992. PMID: 1362901 Free PMC article.

3

Rsr1 and Rap1 GTPases are activated by the same GTPase-activating protein and require threonine 65 for their activation.

Holden JL, Nur-E-Kamal MS, Fabri L, Nice E, Hammacher A, Maruta H. Holden JL, et al. Among authors: maruta h. J Biol Chem. 1991 Sep 15;266(26):16992-5. J Biol Chem. 1991. PMID: 1910037 Free article.

4

rho GAP of 28 kDa (GAP2), but not of 190 kDa (p190), requires Asp65 and Asp67 of rho GTPase for its activation.

Morii N, Kumagai N, Nur-E-Kamal MS, Narumiya S, Maruta H. Morii N, et al. Among authors: maruta h. J Biol Chem. 1993 Dec 25;268(36):27160-3. J Biol Chem. 1993. PMID: 7903304 Free article.

5

The GTPase-activating NF1 fragment of 91 amino acids reverses v-Ha-Ras-induced malignant phenotype.

Nur-E-Kamal MS, Varga M, Maruta H. Nur-E-Kamal MS, et al. Among authors: maruta h. J Biol Chem. 1993 Oct 25;268(30):22331-7. J Biol Chem. 1993. PMID: 8226742 Free article.

6

Asparagine 26, glutamic acid 31, valine 45, and tyrosine 64 of Ras proteins are required for their oncogenicity.

Nur-E-Kamal MS, Sizeland A, D'Abaco G, Maruta H. Nur-E-Kamal MS, et al. Among authors: maruta h. J Biol Chem. 1992 Jan 25;267(3):1415-8. J Biol Chem. 1992. PMID: 1730690 Free article.

7

The GTPase and Rho GAP domains of p190, a tumor suppressor protein that binds the M(r) 120,000 Ras GAP, independently function as anti-Ras tumor suppressors.

Wang DZ, Nur-E-Kamal MS, Tikoo A, Montague W, Maruta H. Wang DZ, et al. Among authors: maruta h. Cancer Res. 1997 Jun 15;57(12):2478-84. Cancer Res. 1997. PMID: 9192829

8

The CDC42-specific inhibitor derived from ACK-1 blocks v-Ha-Ras-induced transformation.

Nur-E-Kamal MS, Kamal JM, Qureshi MM, Maruta H. Nur-E-Kamal MS, et al. Among authors: maruta h. Oncogene. 1999 Dec 16;18(54):7787-93. doi: 10.1038/sj.onc.1203215. Oncogene. 1999. PMID: 10618719

9

Construction of a cell-permeable CDC42 binding fragment of ACK that inhibits v-Ha-Ras transformation.

Nur-e-Kamal MS, Qureshi MM, Kamal JM, Montague W, Maruta H. Nur-e-Kamal MS, et al. Among authors: maruta h. Ann N Y Acad Sci. 1999;886:285-8. doi: 10.1111/j.1749-6632.1999.tb09438.x. Ann N Y Acad Sci. 1999. PMID: 10667241 Review. No abstract available.

10

Midkine (MK), a retinoic acid (RA)-inducible gene product, produced in E. coli acts on neuronal and HL60 leukemia cells.

Maruta H, Bartlett PF, Nurcombe V, Nur-E-Kamal MS, Chomienne C, Muramatsu T, Muramatsu H, Fabri L, Nice E, Burgess AW. Maruta H, et al. Growth Factors. 1993;8(2):119-34. doi: 10.3109/08977199309046932. Growth Factors. 1993. PMID: 8466754

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