Farhood A - Search Results

1

Activation of Kupffer cells and neutrophils for reactive oxygen formation is responsible for endotoxin-enhanced liver injury after hepatic ischemia.

Liu P, McGuire GM, Fisher MA, Farhood A, Smith CW, Jaeschke H. Liu P, et al. Among authors: farhood a. Shock. 1995 Jan;3(1):56-62. Shock. 1995. PMID: 7850581

2

Cytokine-induced upregulation of hepatic intercellular adhesion molecule-1 messenger RNA expression and its role in the pathophysiology of murine endotoxin shock and acute liver failure.

Essani NA, Fisher MA, Farhood A, Manning AM, Smith CW, Jaeschke H. Essani NA, et al. Among authors: farhood a. Hepatology. 1995 Jun;21(6):1632-9. Hepatology. 1995. PMID: 7768509

3

Beneficial effects of extracellular glutathione against endotoxin-induced liver injury during ischemia and reperfusion.

Liu P, Fisher MA, Farhood A, Smith CW, Jaeschke H. Liu P, et al. Among authors: farhood a. Circ Shock. 1994 Jun;43(2):64-70. Circ Shock. 1994. PMID: 7834822

4

The 21-aminosteroid tirilazad mesylate protects against endotoxin shock and acute liver failure in rats.

Liu P, Vonderfecht SL, McGuire GM, Fisher MA, Farhood A, Jaeschke H. Liu P, et al. Among authors: farhood a. J Pharmacol Exp Ther. 1994 Oct;271(1):438-45. J Pharmacol Exp Ther. 1994. PMID: 7965745

5

Inhibition of nitric oxide synthesis aggravates reperfusion injury after hepatic ischemia and endotoxemia.

Wang Y, Mathews WR, Guido DM, Farhood A, Jaeschke H. Wang Y, et al. Among authors: farhood a. Shock. 1995 Oct;4(4):282-8. doi: 10.1097/00024382-199510000-00009. Shock. 1995. PMID: 8564557

6

Release of soluble intercellular adhesion molecule 1 into bile and serum in murine endotoxin shock.

Jaeschke H, Essani NA, Fisher MA, Vonderfecht SL, Farhood A, Smith CW. Jaeschke H, et al. Among authors: farhood a. Hepatology. 1996 Mar;23(3):530-6. doi: 10.1002/hep.510230318. Hepatology. 1996. PMID: 8617433

7

Sequestration of neutrophils in the hepatic vasculature during endotoxemia is independent of beta 2 integrins and intercellular adhesion molecule-1.

Jaeschke H, Farhood A, Fisher MA, Smith CW. Jaeschke H, et al. Among authors: farhood a. Shock. 1996 Nov;6(5):351-6. doi: 10.1097/00024382-199611000-00009. Shock. 1996. PMID: 8946651

8

Increased P-selectin gene expression in the liver vasculature and its role in the pathophysiology of neutrophil-induced liver injury in murine endotoxin shock.

Essani NA, Fisher MA, Simmons CA, Hoover JL, Farhood A, Jaeschke H. Essani NA, et al. Among authors: farhood a. J Leukoc Biol. 1998 Mar;63(3):288-96. doi: 10.1002/jlb.63.3.288. J Leukoc Biol. 1998. PMID: 9500515

9

Parenchymal cell apoptosis as a signal for sinusoidal sequestration and transendothelial migration of neutrophils in murine models of endotoxin and Fas-antibody-induced liver injury.

Lawson JA, Fisher MA, Simmons CA, Farhood A, Jaeschke H. Lawson JA, et al. Among authors: farhood a. Hepatology. 1998 Sep;28(3):761-7. doi: 10.1002/hep.510280324. Hepatology. 1998. PMID: 9731570

10

Pathophysiologic importance of E- and L-selectin for neutrophil-induced liver injury during endotoxemia in mice.

Lawson JA, Burns AR, Farhood A, Lynn Bajt M, Collins RG, Smith CW, Jaeschke H. Lawson JA, et al. Among authors: farhood a. Hepatology. 2000 Nov;32(5):990-8. doi: 10.1053/jhep.2000.19068. Hepatology. 2000. PMID: 11050049

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