Hong WK - Search Results

1

All-trans-retinoic acid increases transforming growth factor-beta2 and insulin-like growth factor binding protein-3 expression through a retinoic acid receptor-alpha-dependent signaling pathway.

Han GR, Dohi DF, Lee HY, Rajah R, Walsh GL, Hong WK, Cohen P, Kurie JM. Han GR, et al. Among authors: hong wk. J Biol Chem. 1997 May 23;272(21):13711-6. doi: 10.1074/jbc.272.21.13711. J Biol Chem. 1997. PMID: 9153223 Free article.

2

Insulin-like growth factor binding protein-3 inhibits the growth of non-small cell lung cancer.

Lee HY, Chun KH, Liu B, Wiehle SA, Cristiano RJ, Hong WK, Cohen P, Kurie JM. Lee HY, et al. Among authors: hong wk. Cancer Res. 2002 Jun 15;62(12):3530-7. Cancer Res. 2002. PMID: 12068000

3

Retinoid refractoriness occurs during lung carcinogenesis despite functional retinoid receptors.

Kim YH, Dohi DF, Han GR, Zou CP, Oridate N, Walsh GL, Nesbitt JC, Xu XC, Hong WK, Lotan R, et al. Kim YH, et al. Among authors: hong wk. Cancer Res. 1995 Dec 1;55(23):5603-10. Cancer Res. 1995. PMID: 7585641

4

Retinoic acid receptor- and retinoid X receptor-selective retinoids activate signaling pathways that converge on AP-1 and inhibit squamous differentiation in human bronchial epithelial cells.

Lee HY, Dawson MI, Walsh GL, Nesbitt JC, Eckert RL, Fuchs E, Hong WK, Lotan R, Kurie JM. Lee HY, et al. Among authors: hong wk. Cell Growth Differ. 1996 Aug;7(8):997-1004. Cell Growth Differ. 1996. PMID: 8853895

5

Evidence of a retinoid signaling alteration involving the activator protein 1 complex in tumorigenic human bronchial epithelial cells and non-small cell lung cancer cells.

Lee HY, Dawson MI, Claret FX, Chen JD, Walsh GL, Hong WK, Kurie JM. Lee HY, et al. Among authors: hong wk. Cell Growth Differ. 1997 Mar;8(3):283-91. Cell Growth Differ. 1997. PMID: 9056670

6

All-trans retinoic acid converts E2F into a transcriptional suppressor and inhibits the growth of normal human bronchial epithelial cells through a retinoic acid receptor- dependent signaling pathway.

Lee HY, Dohi DF, Kim YH, Walsh GL, Consoli U, Andreeff M, Dawson MI, Hong WK, Kurie JM. Lee HY, et al. Among authors: hong wk. J Clin Invest. 1998 Mar 1;101(5):1012-9. doi: 10.1172/JCI1329. J Clin Invest. 1998. PMID: 9486971 Free PMC article.

7

All-trans-retinoic acid inhibits Jun N-terminal kinase-dependent signaling pathways.

Lee HY, Walsh GL, Dawson MI, Hong WK, Kurie JM. Lee HY, et al. Among authors: hong wk. J Biol Chem. 1998 Mar 20;273(12):7066-71. doi: 10.1074/jbc.273.12.7066. J Biol Chem. 1998. PMID: 9507016 Free article.

8

Suppression of c-Fos gene transcription with malignant transformation of human bronchial epithelial cells.

Lee HY, Chaudhary J, Walsh GL, Hong WK, Kurie JM. Lee HY, et al. Among authors: hong wk. Oncogene. 1998 Jun 11;16(23):3039-46. doi: 10.1038/sj.onc.1201843. Oncogene. 1998. PMID: 9662337

9

All-trans-retinoic acid inhibits Jun N-terminal kinase by increasing dual-specificity phosphatase activity.

Lee HY, Sueoka N, Hong WK, Mangelsdorf DJ, Claret FX, Kurie JM. Lee HY, et al. Among authors: hong wk. Mol Cell Biol. 1999 Mar;19(3):1973-80. doi: 10.1128/MCB.19.3.1973. Mol Cell Biol. 1999. PMID: 10022884 Free PMC article.

10

Posttranslational mechanisms contribute to the suppression of specific cyclin:CDK complexes by all-trans retinoic acid in human bronchial epithelial cells.

Sueoka N, Lee HY, Walsh GL, Hong WK, Kurie JM. Sueoka N, et al. Among authors: hong wk. Cancer Res. 1999 Aug 1;59(15):3838-44. Cancer Res. 1999. PMID: 10447003

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