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Somatic and germline mutations of the TSH receptor gene in thyroid diseases.
Van Sande J, Parma J, Tonacchera M, Swillens S, Dumont J, Vassart G. Van Sande J, et al. Among authors: parma j. J Clin Endocrinol Metab. 1995 Sep;80(9):2577-85. doi: 10.1210/jcem.80.9.7673398. J Clin Endocrinol Metab. 1995. PMID: 7673398 Review. No abstract available.
Somatic mutations causing constitutive activity of the thyrotropin receptor are the major cause of hyperfunctioning thyroid adenomas: identification of additional mutations activating both the cyclic adenosine 3',5'-monophosphate and inositol phosphate-Ca2+ cascades.
Parma J, Van Sande J, Swillens S, Tonacchera M, Dumont J, Vassart G. Parma J, et al. Mol Endocrinol. 1995 Jun;9(6):725-33. doi: 10.1210/mend.9.6.8592518. Mol Endocrinol. 1995. PMID: 8592518
The TSH receptor and thyroid diseases.
Paschke R, Van Sande J, Parma J, Vassart G. Paschke R, et al. Among authors: parma j. Baillieres Clin Endocrinol Metab. 1996 Jan;10(1):9-27. doi: 10.1016/s0950-351x(96)80266-7. Baillieres Clin Endocrinol Metab. 1996. PMID: 8734449 Review.
TSH receptor and disease.
Tonacchera M, Van Sande J, Parma J, Duprez L, Cetani F, Costagliola S, Dumont JE, Vassart G. Tonacchera M, et al. Among authors: parma j. Clin Endocrinol (Oxf). 1996 Jun;44(6):621-33. doi: 10.1046/j.1365-2265.1996.746567.x. Clin Endocrinol (Oxf). 1996. PMID: 8759174 Review. No abstract available.
91 results