Bates GP - Search Results

1

Are neuronal intranuclear inclusions the common neuropathology of triplet-repeat disorders with polyglutamine-repeat expansions?

Davies SW, Beardsall K, Turmaine M, DiFiglia M, Aronin N, Bates GP. Davies SW, et al. Among authors: bates gp. Lancet. 1998 Jan 10;351(9096):131-3. doi: 10.1016/S0140-6736(97)08360-8. Lancet. 1998. PMID: 9439509

2

Exon 1 of the HD gene with an expanded CAG repeat is sufficient to cause a progressive neurological phenotype in transgenic mice.

Mangiarini L, Sathasivam K, Seller M, Cozens B, Harper A, Hetherington C, Lawton M, Trottier Y, Lehrach H, Davies SW, Bates GP. Mangiarini L, et al. Among authors: bates gp. Cell. 1996 Nov 1;87(3):493-506. doi: 10.1016/s0092-8674(00)81369-0. Cell. 1996. PMID: 8898202 Free article.

3

Instability of highly expanded CAG repeats in mice transgenic for the Huntington's disease mutation.

Mangiarini L, Sathasivam K, Mahal A, Mott R, Seller M, Bates GP. Mangiarini L, et al. Among authors: bates gp. Nat Genet. 1997 Feb;15(2):197-200. doi: 10.1038/ng0297-197. Nat Genet. 1997. PMID: 9020849

4

Identification of an HD patient with a (CAG)180 repeat expansion and the propagation of highly expanded CAG repeats in lambda phage.

Sathasivam K, Amaechi I, Mangiarini L, Bates G. Sathasivam K, et al. Hum Genet. 1997 May;99(5):692-5. doi: 10.1007/s004390050432. Hum Genet. 1997. PMID: 9150744

5

Formation of neuronal intranuclear inclusions underlies the neurological dysfunction in mice transgenic for the HD mutation.

Davies SW, Turmaine M, Cozens BA, DiFiglia M, Sharp AH, Ross CA, Scherzinger E, Wanker EE, Mangiarini L, Bates GP. Davies SW, et al. Among authors: bates gp. Cell. 1997 Aug 8;90(3):537-48. doi: 10.1016/s0092-8674(00)80513-9. Cell. 1997. PMID: 9267033 Free article.

6

Huntingtin-encoded polyglutamine expansions form amyloid-like protein aggregates in vitro and in vivo.

Scherzinger E, Lurz R, Turmaine M, Mangiarini L, Hollenbach B, Hasenbank R, Bates GP, Davies SW, Lehrach H, Wanker EE. Scherzinger E, et al. Among authors: bates gp. Cell. 1997 Aug 8;90(3):549-58. doi: 10.1016/s0092-8674(00)80514-0. Cell. 1997. PMID: 9267034 Free article.

7

Transgenic models of Huntington's disease.

Bates GP, Mangiarini L, Mahal A, Davies SW. Bates GP, et al. Hum Mol Genet. 1997;6(10):1633-7. doi: 10.1093/hmg/6.10.1633. Hum Mol Genet. 1997. PMID: 9300654 Review.

8

Aggregation of huntingtin in neuronal intranuclear inclusions and dystrophic neurites in brain.

DiFiglia M, Sapp E, Chase KO, Davies SW, Bates GP, Vonsattel JP, Aronin N. DiFiglia M, et al. Among authors: bates gp. Science. 1997 Sep 26;277(5334):1990-3. doi: 10.1126/science.277.5334.1990. Science. 1997. PMID: 9302293

9

Transgenic mouse models of neurodegenerative disease caused by CAG/polyglutamine expansions.

Bates GP, Davies SW. Bates GP, et al. Mol Med Today. 1997 Nov;3(11):508-15. doi: 10.1016/S1357-4310(97)01142-8. Mol Med Today. 1997. PMID: 9430787 Review.

10

HAP1-huntingtin interactions do not contribute to the molecular pathology in Huntington's disease transgenic mice.

Bertaux F, Sharp AH, Ross CA, Lehrach H, Bates GP, Wanker E. Bertaux F, et al. Among authors: bates gp. FEBS Lett. 1998 Apr 17;426(2):229-32. doi: 10.1016/s0014-5793(98)00352-4. FEBS Lett. 1998. PMID: 9599014 Free article.

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