van Rij RP - Search Results

1

Role of CCR2 genotype in the clinical course of syncytium-inducing (SI) or non-SI human immunodeficiency virus type 1 infection and in the time to conversion to SI virus variants.

van Rij RP, de Roda Husman AM, Brouwer M, Goudsmit J, Coutinho RA, Schuitemaker H. van Rij RP, et al. J Infect Dis. 1998 Dec;178(6):1806-11. doi: 10.1086/314522. J Infect Dis. 1998. PMID: 9815240

2

CC chemokine receptor 5 delta32 and CC chemokine receptor 2 64I polymorphisms do not influence the virologic and immunologic response to antiretroviral combination therapy in human immunodeficiency virus type 1-infected patients.

Wit FW, van Rij RP, Weverling GJ, Lange JM, Schuitemaker H. Wit FW, et al. Among authors: van rij rp. J Infect Dis. 2002 Dec 15;186(12):1726-32. doi: 10.1086/345677. Epub 2002 Nov 22. J Infect Dis. 2002. PMID: 12447757

3

The role of a stromal cell-derived factor-1 chemokine gene variant in the clinical course of HIV-1 infection.

van Rij RP, Broersen S, Goudsmit J, Coutinho RA, Schuitemaker H. van Rij RP, et al. AIDS. 1998 Jun 18;12(9):F85-90. AIDS. 1998. PMID: 9662191

4

Reduced prevalence of the CCR5 delta32 heterozygous genotype in human immunodeficiency virus-infected individuals with AIDS dementia complex.

van Rij RP, Portegies P, Hallaby T, Lange JM, Visser J, de Roda Husman AM, van 't Wout AB, Schuitemaker H. van Rij RP, et al. Among authors: van t wout ab. J Infect Dis. 1999 Sep;180(3):854-7. doi: 10.1086/314940. J Infect Dis. 1999. PMID: 10438379

5

Adaptation to promiscuous usage of chemokine receptors is not a prerequisite for human immunodeficiency virus type 1 disease progression.

de Roda Husman AM, van Rij RP, Blaak H, Broersen S, Schuitemaker H. de Roda Husman AM, et al. Among authors: van rij rp. J Infect Dis. 1999 Oct;180(4):1106-15. doi: 10.1086/314987. J Infect Dis. 1999. PMID: 10479137

6

Differential coreceptor expression allows for independent evolution of non-syncytium-inducing and syncytium-inducing HIV-1.

van Rij RP, Blaak H, Visser JA, Brouwer M, Rientsma R, Broersen S, de Roda Husman AM, Schuitemaker H. van Rij RP, et al. J Clin Invest. 2000 Oct;106(8):1039-52. doi: 10.1172/JCI7953. J Clin Invest. 2000. PMID: 11032864 Free PMC article.

7

Both R5 and X4 human immunodeficiency virus type 1 variants persist during prolonged therapy with five antiretroviral drugs.

van Rij RP, Visser JA, van Praag RM, Rientsma R, Prins JM, Lange JM, Schuitemaker H. van Rij RP, et al. Among authors: van praag rm. J Virol. 2002 Mar;76(6):3054-8. doi: 10.1128/jvi.76.6.3054-3058.2002. J Virol. 2002. PMID: 11861873 Free PMC article.

8

Host genetic factors in the clinical course of HIV-1 infection: chemokines and chemokine receptors.

van Rij RP, Schuitemaker H. van Rij RP, et al. Community Genet. 2002;5(2):88-101. doi: 10.1159/000065169. Community Genet. 2002. PMID: 12811024

9

In vivo evolution of X4 human immunodeficiency virus type 1 variants in the natural course of infection coincides with decreasing sensitivity to CXCR4 antagonists.

Stalmeijer EH, Van Rij RP, Boeser-Nunnink B, Visser JA, Naarding MA, Schols D, Schuitemaker H. Stalmeijer EH, et al. Among authors: van rij rp. J Virol. 2004 Mar;78(6):2722-8. doi: 10.1128/jvi.78.6.2722-2728.2004. J Virol. 2004. PMID: 14990692 Free PMC article.

10

Association between an interleukin-4 promoter polymorphism and the acquisition of CXCR4 using HIV-1 variants.

Kwa D, van Rij RP, Boeser-Nunnink B, Vingerhoed J, Schuitemaker H. Kwa D, et al. Among authors: van rij rp. AIDS. 2003 May 2;17(7):981-5. doi: 10.1097/00002030-200305020-00006. AIDS. 2003. PMID: 12700447

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