Neuroprotective effect of ciclopirox olamine in retinal ischemia/reperfusion injury

BMC Mol Cell Biol. 2024 Oct 9;25(1):22. doi: 10.1186/s12860-024-00520-w.

Abstract

Retinal ischemia-reperfusion (IR) injury is a basic pathological procedure in clinic and associated with various ischemic retinal diseases, including glaucoma, diabetic retinopathy, retinal vascular occlusion, etc. The purpose of this work is to investigate the effect of ciclopirox olamine (CPX) on retinal IR injury and further explore the underlying mechanism. In vitro assay exhibited that CPX exhibited significant neuroprotection against oxygen glucose deprivation (OGD) and oxidative stress-induced injuries in 661W photoreceptor cells. OGD injury showed a proinflammatory phenotype characterized by significantly increased production of cytokines (IL-6, IL-23 and TNF-α), while CPX significantly inhibited their secretion. In addition, the in vivo experiment demonstrated that CPX significantly preserved the normal thickness of the retina. Therefore, we suggest that CPX is identified in our research as a prospective therapeutic agent for retinal IR injury.

Keywords: Ciclopirox olamine; Ischemia/reperfusion injury; Neuroprotection; Retina.

MeSH terms

  • Animals
  • Cell Line
  • Ciclopirox* / pharmacology
  • Ciclopirox* / therapeutic use
  • Cytokines / metabolism
  • Glucose / metabolism
  • Male
  • Mice
  • Mice, Inbred C57BL
  • Neuroprotective Agents* / pharmacology
  • Neuroprotective Agents* / therapeutic use
  • Oxidative Stress* / drug effects
  • Photoreceptor Cells, Vertebrate / drug effects
  • Photoreceptor Cells, Vertebrate / metabolism
  • Photoreceptor Cells, Vertebrate / pathology
  • Reperfusion Injury* / drug therapy
  • Reperfusion Injury* / metabolism
  • Retina / drug effects
  • Retina / metabolism
  • Retina / pathology
  • Retinal Diseases / drug therapy
  • Retinal Diseases / metabolism

Substances

  • Ciclopirox
  • Neuroprotective Agents
  • Cytokines
  • Glucose