R01AG075802/AG/NIA NIH HHS/United States[Grants and Funding] - Search Results

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Neuronal and glial vulnerability of the suprachiasmatic nucleus in tauopathies: evidence from human studies and animal models.

Son G, Neylan TC, Grinberg LT. Son G, et al. Mol Neurodegener. 2024 Jan 10;19(1):4. doi: 10.1186/s13024-023-00695-4. Mol Neurodegener. 2024. PMID: 38195580 Free PMC article. Review.

Alzheimer's disease clinical variants show distinct neuroinflammatory profiles with neuropathology.

Boon BDC, Frigerio I, de Gooijer D, Morrema THJ, Bol J, Galis-de Graaf Y, Heymans M; Netherlands Brain Bank; Murray ME; Normal Aging Brain Collection Amsterdam; van der Lee SJ, Holstege H, van de Berg WDJ, Jonkman LE, Rozemuller AJM, Bouwman FH, Hoozemans JJM. Boon BDC, et al. Neuropathol Appl Neurobiol. 2024 Oct;50(5):e13009. doi: 10.1111/nan.13009. Neuropathol Appl Neurobiol. 2024. PMID: 39400356

Exploring the significance of caspase-cleaved tau in tauopathies and as a complementary pathology to phospho-tau in Alzheimer's disease: implications for biomarker development and therapeutic targeting.

Rizzi L, Grinberg LT. Rizzi L, et al. Acta Neuropathol Commun. 2024 Feb 28;12(1):36. doi: 10.1186/s40478-024-01744-9. Acta Neuropathol Commun. 2024. PMID: 38419122 Free PMC article. Review.

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