Oxidative stress and matrix metalloproteinase-9 in acute ischemic stroke: the Biomarker Evaluation for Antioxidant Therapies in Stroke (BEAT-Stroke) study

Stroke. 2008 Jan;39(1):100-4. doi: 10.1161/STROKEAHA.107.488189. Epub 2007 Dec 6.

Abstract

Background and purpose: Experimental stroke studies indicate that oxidative stress is a major contributing factor to ischemic cerebral injury. Oxidative stress is also implicated in activation of matrix metalloproteinases (MMPs) and blood-brain barrier injury after ischemia-reperfusion. Plasma biomarkers of oxidative stress may have utility as early indicators of efficacy in Phase 2 trials of antioxidant therapies in human stroke. To date, a valid biomarker has been unavailable. We measured F2-isoprostanes (F2IPs), free-radical induced products of neuronal arachadonic acid peroxidation, in acute ischemic stroke. We aimed to determine the change in plasma F2IP levels over time and relationship with plasma MMP-9 in tPA-treated and tPA-untreated stroke patients.

Methods: We performed a case-control study of consecutive ischemic stroke patients (25 tPA-treated and 27 tPA-untreated) presenting within 8 hours of stroke onset. Controls were individuals without prior stroke from a primary care clinic network serving the source population from which cases were derived. Infarct volume was determined on acute diffusion-weighted MRI (DWI) performed within 48 hours using a semi-automated computerized segmentation algorithm. Phlebotomy was performed at <8 hours, 24 hours, 2 to 5 days, and 4 to 6 weeks. F2IPs were measured by gas chromatography/mass spectrometry and MMP-9 by ELISA. Prestroke antioxidant dietary intake was measured by the 24-hour recall method.

Results: In 52 cases and 27 controls, early (median 6 hours postonset) F2IPs were elevated in stroke cases compared with controls (medians 0. 041 versus 0.0295 pg/mL, P=0.012). No difference in F2IPSs was present at later time points. Early plasma F2IPs correlated with MMP-9 in all patients (P=0.01) and the tPA-treated subgroup (P=0.02). No correlation was found with NIHSS, DWI infarct volume, 90-day Rankin score, or C-reactive protein (P>0.05 for all).

Conclusions: In early human stroke we found evidence of increased oxidative stress and a relationship with MMP-9 expression, supporting findings from experimental studies.

Publication types

  • Comparative Study
  • Research Support, N.I.H., Extramural
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Aged
  • Aged, 80 and over
  • Antioxidants / pharmacology
  • Ascorbic Acid / pharmacology
  • Biomarkers / blood
  • Brain Ischemia / blood*
  • Brain Ischemia / drug therapy
  • Brain Ischemia / physiopathology
  • Case-Control Studies
  • Cerebral Infarction / pathology
  • Diffusion Magnetic Resonance Imaging
  • F2-Isoprostanes / blood*
  • Female
  • Fibrinolytic Agents / therapeutic use
  • Humans
  • Male
  • Matrix Metalloproteinase 9 / blood*
  • Middle Aged
  • Oxidative Stress / physiology*
  • Severity of Illness Index
  • Stroke / blood*
  • Stroke / drug therapy
  • Stroke / physiopathology
  • Tissue Plasminogen Activator / therapeutic use
  • Treatment Outcome
  • alpha-Tocopherol / pharmacology

Substances

  • Antioxidants
  • Biomarkers
  • F2-Isoprostanes
  • Fibrinolytic Agents
  • Tissue Plasminogen Activator
  • Matrix Metalloproteinase 9
  • alpha-Tocopherol
  • Ascorbic Acid