Expression of Fas-related genes in human hepatocellular carcinomas

Cancer Lett. 1998 Dec 25;134(2):155-62. doi: 10.1016/s0304-3835(98)00251-1.

Abstract

Hepatocellular carcinoma (HCC) is one of the tumors known to be resistant to Fas-mediated apoptosis. To elucidate the possible mechanisms of this resistance, we examined the mRNA transcripts of genes related to Fas in 10 hepatitis B virus (HBV)-associated HCC cell lines and six HBV-associated HCC tissues. Most of the HBV-associated HCC cell lines showed markedly decreased Fas expression. Some cell lines expressed FAP, a possible inhibitor of apoptosis, or showed downregulated expression of FADD or FLICE, molecules essential to Fas-mediated apoptosis. All six HBV-associated HCC tissues also showed decreased Fas expression compared with their non-malignant counterparts. Our data demonstrate that HBV-associated HCC showed a common defect in the expression of Fas, upregulation of FAP and downregulation of downstream molecules such as FADD and FLICE.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Apoptosis / genetics*
  • Carcinoma, Hepatocellular / genetics*
  • Carcinoma, Hepatocellular / virology
  • Down-Regulation
  • Flow Cytometry
  • Fluorescent Antibody Technique
  • Gene Expression Regulation, Neoplastic*
  • Hepatitis B / complications
  • Humans
  • Liver Neoplasms / genetics*
  • Liver Neoplasms / virology
  • Proto-Oncogene Proteins / genetics*
  • RNA, Messenger / analysis
  • RNA, Neoplasm / analysis
  • Tumor Cells, Cultured
  • Up-Regulation

Substances

  • Proto-Oncogene Proteins
  • RNA, Messenger
  • RNA, Neoplasm