Increase in IL-8, IL-10, IL-13, and RANTES mRNA levels (in situ hybridization) in the nasal mucosa after nasal allergen provocation

J Allergy Clin Immunol. 1999 Mar;103(3 Pt 1):441-50. doi: 10.1016/s0091-6749(99)70469-0.

Abstract

Background: Allergic inflammation is regulated by the local production and release of several cytokines.

Objectives: This study was designed to assess the changes in mRNA cytokine-positive cells after allergen provocation and to compare these cytokines with tissue eosinophilia as a marker of allergic inflammation.

Methods: A grass pollen allergen provocation study was conducted in autumn, out of the hay fever season. Nasal mucosal biopsy specimens were taken before provocation and 1 hour, 24 hours, and 1 week after allergen provocation. Eosinophils and mRNA-positive cells (in situ hybridization for IL-2, IL-3, IL-4, IL-5, IL-6, IL-8, IL-10, IL-13, IFN-gamma, RANTES, and TNF-alpha) were assessed in the biopsy specimens.

Results: After allergen provocation, an increase in cell number was found for eosinophils and cells expressing mRNA for the chemokines IL-8 and RANTES and for the TH2 cytokines IL-10 and IL-13. Significant correlations were found between eosinophils and RANTES and eosinophils and IFN-gamma in the early phase and between eosinophils and IL-5 and eosinophils and RANTES in the late phase. The increase in eosinophils and IL-10 and IL-13 mRNA-positive cells could still be observed 1 week after allergen provocation.

Conclusions: Nasal allergen provocation induced significant tissue eosinophilia and a significant increase in IL-8, IL-13, and RANTES mRNA-positive cells. A significant increase in eosinophils and IL-10 and IL-13 mRNA-positive cells compared with baseline can still be observed 1 week after a single allergen provocation.

MeSH terms

  • Adult
  • Allergens*
  • Biopsy
  • Cell Count
  • Chemokine CCL5 / biosynthesis
  • Chemokine CCL5 / genetics*
  • Eosinophilia / etiology*
  • Eosinophilia / genetics
  • Eosinophilia / metabolism
  • Eosinophils
  • Female
  • Humans
  • In Situ Hybridization
  • Interferon-gamma / genetics
  • Interleukin-10 / biosynthesis
  • Interleukin-10 / genetics*
  • Interleukin-13 / biosynthesis
  • Interleukin-13 / genetics*
  • Interleukin-8 / biosynthesis
  • Interleukin-8 / genetics*
  • Interleukins / genetics
  • Male
  • Middle Aged
  • Nasal Mucosa / metabolism*
  • Nasal Mucosa / pathology
  • Nasal Provocation Tests*
  • Poaceae
  • Pollen / immunology*
  • RNA, Messenger / biosynthesis*
  • Rhinitis, Allergic, Seasonal / genetics
  • Rhinitis, Allergic, Seasonal / metabolism*
  • Rhinitis, Allergic, Seasonal / pathology
  • Seasons
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Allergens
  • Chemokine CCL5
  • Interleukin-13
  • Interleukin-8
  • Interleukins
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • Interleukin-10
  • Interferon-gamma