Persistent alterations in T-cell repertoire, cytokine and chemokine receptor gene expression after 1 year of highly active antiretroviral therapy

AIDS. 1999 Feb 4;13(2):185-94. doi: 10.1097/00002030-199902040-00006.

Abstract

Objectives: To examine T-cell repertoire modifications, the evolution of T-helper (TH)1/TH2 cytokine imbalance and modifications in chemokine receptor expression when the viral load is decreased by 2-3 log10 copies/ml under highly active antiretroviral therapy (HAART).

Design: Sixteen patients previously treated with zidovudine and lamivudine, with CD4 cells below 300 x 10(6)/l and viraemia above 30000 copies/ml were treated by saquinavir and ritonavir together with both reverse transcriptase (RT) inhibitors (ANRS 069 trial). T-cell repertoire, chemokine receptor and lymphokine expression were studied from peripheral blood mononuclear cells sampled at weeks 0, 24 and 48.

Methods: T-cell repertoire study was carried out using the Immunoscope method. Interleukin (IL)-12 receptor beta2, CC-chemokine receptor (CCR)-3, CXC-chemokine receptor-4 and CCR-5 expression in CD4+ cells was measured by kinetic quantitative PCR and IL-2, IL-4, IL-10, IL-13, interferon (IFN)-gamma were measured using a quantitative RT-PCR assay with homologous internal standards.

Results: Repertoire alterations were more frequent in CD4- than in CD4+ cells and persisted despite undetectable viraemia. Increased CCR-3 expression and spontaneous IFN-gamma as well as mitogenic induced IL-13 were observed at baseline and decreased slightly under HAART.

Conclusion: The CD8+ cell repertoire alterations were profound, whereas the CD4+ cell alterations were moderate and both persisted unchanged under HAART. The TH1/TH2 imbalance was more related to TH2 over-expression than to TH1 deficiency and persisted for at least 1 year under HAART.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Anti-HIV Agents / therapeutic use*
  • CD4-Positive T-Lymphocytes / immunology*
  • Cytokines / genetics*
  • Female
  • Gene Expression
  • HIV Infections / drug therapy
  • HIV Infections / genetics
  • HIV Infections / immunology*
  • HIV Protease Inhibitors / therapeutic use*
  • Humans
  • Interferon-gamma / genetics
  • Interleukin-10 / genetics
  • Interleukin-13 / genetics
  • Interleukin-2 / genetics
  • Interleukin-4 / genetics
  • Lamivudine / therapeutic use
  • Longitudinal Studies
  • Male
  • RNA, Messenger
  • Receptors, CCR3
  • Receptors, CCR5 / genetics
  • Receptors, CXCR4 / genetics
  • Receptors, Chemokine / genetics*
  • Receptors, Interleukin / genetics
  • Receptors, Interleukin-12
  • Reverse Transcriptase Inhibitors / therapeutic use*
  • Ritonavir / therapeutic use
  • Saquinavir / therapeutic use
  • Zidovudine / therapeutic use

Substances

  • Anti-HIV Agents
  • CCR3 protein, human
  • Cytokines
  • HIV Protease Inhibitors
  • IL12RB2 protein, human
  • Interleukin-13
  • Interleukin-2
  • RNA, Messenger
  • Receptors, CCR3
  • Receptors, CCR5
  • Receptors, CXCR4
  • Receptors, Chemokine
  • Receptors, Interleukin
  • Receptors, Interleukin-12
  • Reverse Transcriptase Inhibitors
  • Interleukin-10
  • Interleukin-4
  • Lamivudine
  • Zidovudine
  • Interferon-gamma
  • Saquinavir
  • Ritonavir