PAI-1 produced ex vivo by human adipose tissue is relevant to PAI-1 blood level

Arterioscler Thromb Vasc Biol. 1999 May;19(5):1361-5. doi: 10.1161/01.atv.19.5.1361.

Abstract

Human adipose tissue has been shown to produce plasminogen activator inhibitor type 1 (PAI-1). However, the importance of adipose tissue in the regulation of the PAI-1 plasma level is not known. The aim of this study was to investigate the relation between the production of PAI-1 by adipose tissue, plasma PAI-1 level, and variables related to the insulin resistance state. The link between the production of PAI-1 inducers such as tumor necrosis factor-alpha and transforming growth factor-beta and the production of PAI-1 by adipose tissue was also evaluated. Blood samples were obtained as soon as possible to the induction of anesthesia from 30 patients undergoing elective abdominoplasty. PAI-1 antigen levels measured in conditioned media after a 19-hour incubation period of adipose tissue explants were significantly correlated with plasma PAI-1 antigen levels (r=0.54, P=0.004) and with systemic lipid parameters such as triglycerides and high density lipoprotein cholesterol (r=0. 46, P=0.014; r=-0.50, P=0.01, respectively) but not with insulinemia and body mass index. PAI-1 production by adipose tissue was correlated with those of TNF-alpha (r=0.5, P=0.01) and TGF-beta (r=0. 53, P=0.007). These results emphasize the role of adipose tissue in determining plasma levels of PAI-1, with a local contribution of TNF-alpha and TGF-beta in PAI-1 production by adipose tissue.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adipose Tissue / metabolism*
  • Adolescent
  • Adult
  • Aged
  • Body Mass Index
  • Cholesterol, HDL / blood
  • Culture Media, Conditioned / chemistry
  • Female
  • Gene Expression Regulation
  • Humans
  • Insulin / blood
  • Insulin Resistance
  • Male
  • Middle Aged
  • Obesity / metabolism*
  • Obesity / pathology
  • Omentum / pathology
  • Organ Culture Techniques
  • Organ Specificity
  • Plasminogen Activator Inhibitor 1 / biosynthesis*
  • Plasminogen Activator Inhibitor 1 / blood
  • Skin / pathology
  • Transforming Growth Factor beta / biosynthesis
  • Triglycerides / blood
  • Tumor Necrosis Factor-alpha / biosynthesis
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Cholesterol, HDL
  • Culture Media, Conditioned
  • Insulin
  • Plasminogen Activator Inhibitor 1
  • Transforming Growth Factor beta
  • Triglycerides
  • Tumor Necrosis Factor-alpha