MCP-1 is selectively expressed in the late phase by cytokine-stimulated human neutrophils: TNF-alpha plays a role in maximal MCP-1 mRNA expression

J Leukoc Biol. 1999 May;65(5):671-9. doi: 10.1002/jlb.65.5.671.

Abstract

Culture supernatants of phytohemagglutinin (PHA)-stimulated human peripheral blood mononuclear cells (PHA-sup) induced monocyte chemoattractant protein-1 (MCP-1) mRNA expression in human neutrophils. MCP-1 mRNA was first detected by Northern analysis at 8 h, and the peak level was detected at 16 h and sustained until 72 h. Cycloheximide and genistein, but not pertussis toxin, inhibited the expression of MCP-1 mRNA. Recombinant tumor necrosis factor alpha (TNF-alpha) induced a low level MCP-1 mRNA accumulation in neutrophils, and addition of anti-TNF-alpha IgG blocked 30-70% of MCP-1 mRNA expression induced with PHA-sup. PHA-sup-stimulated PMN synthesized and secreted 3.1+/-1.3 ng/5 x 10(6) PMN MCP-1 within the first 24 h. Hybridization of 32P-labeled cDNA preparations to an array of human cytokine cDNAs further indicated that MCP-1 mRNA was selectively up-regulated in the late phase after stimulation with the PHA-sup.

MeSH terms

  • Chemokine CCL2 / biosynthesis*
  • Chemokine CCL2 / genetics*
  • Cycloheximide / pharmacology
  • Cytokines / pharmacology*
  • DNA, Complementary / genetics
  • DNA, Complementary / metabolism
  • Genistein / pharmacology
  • Growth Inhibitors / pharmacology
  • Humans
  • Kinetics
  • Neutrophils / drug effects
  • Neutrophils / metabolism*
  • Pertussis Toxin
  • Phytohemagglutinins / pharmacology
  • Protein Synthesis Inhibitors / pharmacology
  • RNA, Messenger / biosynthesis*
  • RNA, Messenger / metabolism
  • Tumor Necrosis Factor-alpha / physiology*
  • Up-Regulation / immunology
  • Virulence Factors, Bordetella / pharmacology

Substances

  • Chemokine CCL2
  • Cytokines
  • DNA, Complementary
  • Growth Inhibitors
  • Phytohemagglutinins
  • Protein Synthesis Inhibitors
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • Virulence Factors, Bordetella
  • Cycloheximide
  • Genistein
  • Pertussis Toxin