Effect of melatonin on cadmium-induced hepatotoxicity in male Sprague-Dawley rats

Tohoku J Exp Med. 1998 Nov;186(3):205-13. doi: 10.1620/tjem.186.205.

Abstract

Effect of melatonin on toxicity of cadmium (Cd) was studied in male SD rats co-administered daily Cd (1 mg/kg b.w., s.c.) with melatonin (10 mg/kg b.w., i.p.) for 15 days. Cd alone injection decreased GSH concentrations in the liver and RBC by 35% and 43% compared with those in saline-treatment group, but not in the kidney and whole brain. The activity of GSSG-reductase was significantly decreased in the liver of Cd alone injected rats, while melatonin given in combination with Cd failed to prevent the Cd-induced decreased activity of hepatic GSSG-reductase. However, the hepatic GSH concentration decreased by Cd alone was restored by melatonin treatment, and the melatonin also ameliorated Cd-induced histopathological changes in the liver. Therefore, data indicate that melatonin restores the reduction of hepatic GSH level induced with Cd regardless of GSSG-reductase activity, and suggests that melatonin may ameliorate Cd-induced hepatotoxicity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Antioxidants / pharmacology*
  • Brain / drug effects
  • Brain / metabolism
  • Cadmium Chloride / toxicity*
  • Drug Combinations
  • Erythrocytes / drug effects
  • Erythrocytes / metabolism
  • Glutathione / metabolism
  • Glutathione Reductase / metabolism
  • Kidney / drug effects
  • Kidney / metabolism
  • Liver / drug effects*
  • Liver / metabolism
  • Liver / pathology
  • Male
  • Melatonin / pharmacology*
  • Rats
  • Rats, Sprague-Dawley

Substances

  • Antioxidants
  • Drug Combinations
  • Glutathione Reductase
  • Glutathione
  • Cadmium Chloride
  • Melatonin