Investigations on the role of cytochrome b5 and divalent cations in the maximal nifedipine oxidase activity of human liver

Arzneimittelforschung. 1999 May;49(5):470-5. doi: 10.1055/s-0031-1300444.

Abstract

Native human cytochrome P4503A4 was most active in nifedipine oxidation when incorporated into a binary phospholipid vesicular system with human NADPH-cytochrome P450 reductase. The turnover numbers were estimated to be 17.6 and 19.6 min-1 in the presence of Mg2+ or Ca2+ ions (5 mmol/l) in the test system, respectively. Inclusion of b5 in the vesicular CYP3A4: reductase system results in a slightly lower nifedipine oxidase activity of 16.9 min-1 in the presence of Mg2+ ions. These results demonstrate that b5 is not an essential component in CYP3A4 catalyzed nifedipine oxidation in human liver.

MeSH terms

  • Amino Acid Sequence
  • Animals
  • Calcium / pharmacology
  • Cations, Divalent / pharmacology*
  • Cholates / metabolism
  • Chromatography, High Pressure Liquid
  • Cytochrome P-450 CYP3A
  • Cytochrome P-450 Enzyme System / metabolism*
  • Cytochromes b5 / metabolism*
  • Glutathione / metabolism
  • Humans
  • In Vitro Techniques
  • Liver / drug effects
  • Liver / enzymology*
  • Magnesium / metabolism
  • Magnesium / pharmacology
  • Mixed Function Oxygenases / metabolism*
  • Molecular Sequence Data
  • NADPH-Ferrihemoprotein Reductase / metabolism
  • Phospholipids / metabolism
  • Rabbits
  • Rats

Substances

  • Cations, Divalent
  • Cholates
  • Phospholipids
  • Cytochromes b5
  • Cytochrome P-450 Enzyme System
  • Mixed Function Oxygenases
  • CYP3A protein, human
  • Cytochrome P-450 CYP3A
  • CYP3A4 protein, human
  • NADPH-Ferrihemoprotein Reductase
  • Glutathione
  • Magnesium
  • Calcium