In vitro production of cytokines is influenced by sulfatide and its precursor galactosylceramide

FEBS Lett. 1999 Jul 23;455(3):339-43. doi: 10.1016/s0014-5793(99)00908-4.

Abstract

Effects of sulfatide and its precursor galactosylceramide (gal-cer) on the kinetics of production of cytokines were studied. In human mononuclear leucocytes, gal-cer but not sulfatide induced significantly increased amounts of interleukin (IL)-1beta, IL-6 and tumor necrosis factor (TNF) mRNA. In phytohemagglutinin-stimulated cultures, gal-cer increased the levels of IL-1beta and IL-6 mRNA and secreted IL-1beta and IL-6, while sulfatide decreased the amounts of IL-6 mRNA and secreted IL-6. Gal-cer also increased TNF secretion. In lipopolysaccharide-stimulated cells, sulfatide but not gal-cer decreased the secretion of IL-1beta and IL-10, a potent suppressor of production of many cytokines. Thus, sulfatide and gal-cer affect cytokine production differently, most likely at the level of gene expression. This may have implications in diseases where inflammatory cytokines play a pathogenic role.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adult
  • Cytokines / biosynthesis*
  • Cytokines / genetics
  • Galactosylceramides / pharmacology*
  • Humans
  • In Vitro Techniques
  • Inflammation Mediators / metabolism
  • Interleukin-1 / biosynthesis
  • Interleukin-1 / genetics
  • Interleukin-6 / biosynthesis
  • Interleukin-6 / genetics
  • Leukocytes, Mononuclear / drug effects
  • Leukocytes, Mononuclear / immunology
  • Leukocytes, Mononuclear / metabolism
  • Lipopolysaccharides / pharmacology
  • Phytohemagglutinins / pharmacology
  • RNA, Messenger / genetics
  • RNA, Messenger / metabolism
  • Sulfoglycosphingolipids / pharmacology*
  • Tumor Necrosis Factor-alpha / biosynthesis
  • Tumor Necrosis Factor-alpha / genetics

Substances

  • Cytokines
  • Galactosylceramides
  • Inflammation Mediators
  • Interleukin-1
  • Interleukin-6
  • Lipopolysaccharides
  • Phytohemagglutinins
  • RNA, Messenger
  • Sulfoglycosphingolipids
  • Tumor Necrosis Factor-alpha