Active site titration of gramicidin S synthetase 2: evidence for misactivation and editing in non-ribosomal peptide biosynthesis

FEBS Lett. 1999 Nov 19;461(3):145-8. doi: 10.1016/s0014-5793(99)01445-3.

Abstract

The catalytic competence of gramicidin S synthetase 2 (GS2) was determined by following the kinetics of PP(i) generation using active site titration measurements with [gamma-(32)P]ATP. The initial 'burst' of product formation can be correlated to the generation of the aminoacyl adenylate:enzyme complexes at the four amino acid activation domains and the subsequent aminoacylation of carrier domains, followed by a slow linear turnover of substrate due to breakdown of the intermediate. Simultaneous activation of all four amino acid substrates at a saturating concentration displayed a consumption of 8.3 ATP/GS2. In the presence of single amino acids, a binding stoichiometry higher than the anticipated two ATP per active site was obtained, implying misactivation at non-cognate domains. Breakdown of acyladenylate intermediates reflects a possible corrective mechanism by which the enzyme controls the fidelity of product formation.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenosine Triphosphate / metabolism
  • Amino Acid Isomerases / metabolism*
  • Binding Sites
  • Catalysis
  • Diphosphates / metabolism
  • Multienzyme Complexes / metabolism*
  • Peptide Biosynthesis*
  • Peptide Synthases / metabolism*

Substances

  • Diphosphates
  • Multienzyme Complexes
  • Adenosine Triphosphate
  • Amino Acid Isomerases
  • Peptide Synthases
  • gramicidin-S-synthetase 2