Abstract
Destruction of IkappaB by ubiquitinylation is required for signal-dependent activation of NF-kappaB. The IkappaB alpha ubiquitin-ligase activity associated with phosphorylated IkappaB alpha (pIkappaB alpha) in HeLa cells was almost completely lost by washing under stringent conditions including 1 M NaCl; nevertheless, an SCF(betaTrCP) complex containing Skp1, Cullin-1, and F-box/WD40 protein betaTrCP was still bound to pIkappaB alpha, suggesting the existence of a putative factor that is loosely associated with pIkappaB alpha and may collaborate with SCF(betaTrCP). The factor was named IkappaB alphaE3-F1 and was partially purified from HeLa cells. Gel filtration analysis revealed that IkappaB alphaE3-F1 has an apparent molecular mass of approximately 300 kDa.
MeSH terms
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Blotting, Western
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Cell Cycle Proteins / metabolism
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Cullin Proteins*
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DNA-Binding Proteins / chemistry*
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DNA-Binding Proteins / metabolism*
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HeLa Cells
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Humans
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I-kappa B Proteins*
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NF-KappaB Inhibitor alpha
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Neoplasm Proteins / chemistry
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Peptide Synthases / metabolism
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Phosphorylation
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Precipitin Tests
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Protein Binding
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S-Phase Kinase-Associated Proteins
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SKP Cullin F-Box Protein Ligases
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Salts / pharmacology
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Tumor Necrosis Factor-alpha / pharmacology
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Ubiquitins / metabolism*
Substances
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Cell Cycle Proteins
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Cullin 1
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Cullin Proteins
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DNA-Binding Proteins
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I-kappa B Proteins
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NFKBIA protein, human
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Neoplasm Proteins
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S-Phase Kinase-Associated Proteins
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Salts
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Tumor Necrosis Factor-alpha
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Ubiquitins
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NF-KappaB Inhibitor alpha
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SKP Cullin F-Box Protein Ligases
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Peptide Synthases