Influence of low dose rIL-2 treatment on endogenous cytokine production, expression of surface IL-2R and the level of soluble IL-2R in patients with minimal residual disease

Leuk Lymphoma. 1999 Oct;35(3-4):355-66. doi: 10.3109/10428199909145740.

Abstract

This study was designed to investigate the immunomodulatory effect of low-dose IL-2 therapy (100 microg/day for 3 weeks) on interferon (IFN), tumor necrosis factor (TNF) production in vivo and in vitro and on the expression of IL-2Ralpha/beta and soluble form of IL-2Ralpha. Patients enrolled in the study suffered from multiple myeloma (MM), Hodgkin's disease (HD) and non-Hodgkin's lymphoma (NHL) All of them were in remission after chemotherapy or radiotherapy. Our results indicated that IL-2 given subcutaneously at a low dose of 100 microg/day for 3 weeks induced IFN-gamma and TNF-alpha in plasma (measured 24 hrs after the last dose of IL-2) and affected the ability of blood leukocytes to produce cytokines. Production of IFN-gamma induced in vitro with PHA was enhanced, but TNF-alpha production induced by lipopolysaccharide (LPS) and virus (Newcastle Disease Virus) was depressed. The expression of both: surface IL-2R, especially beta subunit on total population of lymphocytes and NK cells, and soluble form of IL-2R, of chain were significantly enhanced after low-dose IL-2 therapy. Low dose IL-2 therapy was well tolerated by all patients, and side effects not exceeding II grade of toxicity according to WHO scale were observed. Five patients with MM relapsed 3-10 month after cessation of IL-2 therapy, but all patients with Hodgkin's and non-Hodgkin's lymphomas are still in remission (20 months of observation).

MeSH terms

  • Adult
  • Female
  • Hematologic Neoplasms* / blood
  • Hematologic Neoplasms* / drug therapy
  • Hematologic Neoplasms* / immunology
  • Hematologic Neoplasms* / pathology
  • Humans
  • Interferons / blood
  • Interleukin-2 / administration & dosage*
  • Interleukin-2 / blood*
  • Male
  • Middle Aged
  • Neoplasm, Residual* / blood
  • Neoplasm, Residual* / drug therapy
  • Neoplasm, Residual* / immunology
  • Receptors, Interleukin-2 / blood*
  • Recombinant Proteins / administration & dosage
  • Treatment Outcome
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • Interleukin-2
  • Receptors, Interleukin-2
  • Recombinant Proteins
  • Tumor Necrosis Factor-alpha
  • Interferons