Lack of relationship between plasma thrombomodulin and portal hypertension in alcoholic liver disease

Alcohol. 2000 Feb;20(2):205-6. doi: 10.1016/s0741-8329(99)00072-5.

Abstract

The present study was performed to analyze the relationship between portal hypertension and alterations of the endothelium-derived proteins thrombomodulin, plasminogen activator (t-PA) and plasminogen activator inhibitor (PAI-1), which were determined in plasma samples of 28 alcoholic cirrhotic patients and 46 controls. Although cirrhotics showed lower levels of PAI-1, but higher thrombomodulin and t-PA levels than controls, no relationship was observed between thrombomodulin, t-PA or PAI-1 and portal pressure. Therefore, the hypothesis that splachnic endothelial damage secondary to portal hypertension leads to altered thrombomodulin, t-PA and PAI-1 levels in alcoholic cirrhosis is not supported by the results of this study.

Publication types

  • Clinical Trial

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Endothelium, Vascular / metabolism
  • Female
  • Humans
  • Hypertension, Portal / blood*
  • Liver Diseases, Alcoholic / blood*
  • Male
  • Middle Aged
  • Plasminogen Activator Inhibitor 1 / metabolism
  • Thrombomodulin / blood*
  • Tissue Plasminogen Activator / metabolism

Substances

  • Plasminogen Activator Inhibitor 1
  • Thrombomodulin
  • Tissue Plasminogen Activator