Induction of a novel cellular homolog of interleukin-10, AK155, by transformation of T lymphocytes with herpesvirus saimiri

J Virol. 2000 Apr;74(8):3881-7. doi: 10.1128/jvi.74.8.3881-3887.2000.

Abstract

Although herpesvirus saimiri-transformed T lymphocytes retain multiple normal T-cell functions, only a few changes have been described. By subtractive hybridization, we have isolated a novel cellular gene, ak155, a sequence homolog of the interleukin-10 gene. Specifically herpesvirus saimiri-transformed T cells overexpress ak155 and secrete the protein into the supernatant. In other T-cell lines and in native peripheral blood cells, but not in B cells, ak155 is transcribed at low levels. AK155 forms homodimers similarly to interleukin-10. As a lymphokine, AK155 may contribute to the transformed phenotype of human T cells after infection by herpesvirus saimiri.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Amino Acid Sequence
  • Cell Line, Transformed
  • Cell Transformation, Viral*
  • Cloning, Molecular
  • Herpesvirus 2, Saimiriine / physiology*
  • Humans
  • Interleukin-10 / chemistry
  • Interleukin-10 / genetics*
  • Interleukin-10 / metabolism*
  • Interleukins*
  • Lymphocyte Activation
  • Molecular Sequence Data
  • Nucleic Acid Hybridization
  • Sequence Alignment
  • Sequence Homology, Amino Acid
  • T-Lymphocytes / immunology
  • T-Lymphocytes / virology*
  • Transcription, Genetic

Substances

  • IL26 protein, human
  • Interleukins
  • Interleukin-10

Associated data

  • GENBANK/AJ251549
  • GENBANK/AJ251551