Transforming growth factor-alpha induces the differentiation of sarcomatoid cholangiocarcinoma cells

Jpn J Cancer Res. 2000 Feb;91(2):223-30. doi: 10.1111/j.1349-7006.2000.tb00935.x.

Abstract

A sarcomatoid cholangiocarcinoma cell line, ETK-1, was established from a patient. Phenotypically, the cells corresponded to immature biliary epithelial cells. Because a small number of ETK-1 cells appeared to differentiate spontaneously along a biliary epithelial lineage in continuous culture, we examined the factors that initiate and/or promote the differentiation of the cells. Transforming growth factor-alpha (TGFalpha) induced significant changes in ETK-1 cells. After stimulation with the factor, ETK-1 cells displayed morphologic transformation at a much higher frequency, with the appearance of many large cells with intracytoplasmic vacuoles, and the production of mucinous substances. These morphologically transformed cells were phenotypically similar to well-differentiated adenocarcinoma cells. The expression pattern of integrins after TGFalpha treatment also supported the maturation of the ETK-1 cells. The antibody against the receptor of TGFalpha inhibited these changes by TGFalpha. Moreover, the proliferation rate of ETK-1 cells was suppressed by TGFalpha. Our data suggest that TGFalpha can act as a differentiation factor along a biliary epithelial lineage.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Bile Duct Neoplasms / pathology*
  • Bile Ducts, Intrahepatic*
  • Cell Differentiation / drug effects
  • Cholangiocarcinoma / pathology*
  • Fibroblast Growth Factor 1 / pharmacology
  • Humans
  • Integrins / analysis
  • Proto-Oncogene Proteins c-kit / analysis
  • Receptors, Fibroblast Growth Factor / analysis
  • Transforming Growth Factor alpha / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Integrins
  • Receptors, Fibroblast Growth Factor
  • Transforming Growth Factor alpha
  • Fibroblast Growth Factor 1
  • Proto-Oncogene Proteins c-kit