Destabilization of tumor necrosis factor-alpha mRNA by 5-alpha dihydrotestosterone in Jurkat cells

Life Sci. 2000 Apr 7;66(20):PL277-82. doi: 10.1016/s0024-3205(00)00526-9.

Abstract

The effect of a male steroid hormone, 5DHT, on the expression of TNF-alpha was examined using a human leukemia T cell line, Jurkat. Cells were treated with 5DHT in the presence or absence of PHA, and RNA was isolated followed by a reverse transcriptase - mediated PCR (RT-PCR) to measure the steady state levels of TNF-alpha mRNA. The treatment of cells with 5DHT resulted in a 50% of decrease in the level of TNF-alpha mRNA compared to that in untreated conditions (basal level). A similar level of reduction of the message by 5DHT was also observed in PHA-stimulated cells. The reduction of the steady state levels of TNF-alpha mRNA in Jurkat cells was a result of destabilization of the gene as demonstrated by actinomycin D treatment; a half-life of TNF-alpha message in 5DHT treated cells and non-treated cells was 1 hr and 2.5 hr, respectively, whereas that in 5DHT/PHA and PHA-treated cells was 3hr and 6hr, respectively.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • DNA Primers / chemistry
  • Dactinomycin / pharmacology
  • Dihydrotestosterone / pharmacology*
  • Gene Expression / drug effects
  • Humans
  • Jurkat Cells / drug effects
  • Jurkat Cells / metabolism*
  • Phytohemagglutinins / pharmacology
  • RNA, Messenger / metabolism*
  • RNA, Neoplasm / analysis
  • Reverse Transcriptase Polymerase Chain Reaction
  • Tumor Necrosis Factor-alpha / genetics*
  • Tumor Necrosis Factor-alpha / metabolism

Substances

  • DNA Primers
  • Phytohemagglutinins
  • RNA, Messenger
  • RNA, Neoplasm
  • Tumor Necrosis Factor-alpha
  • Dihydrotestosterone
  • Dactinomycin