Suramin potently inhibits the enzymatic activity of PSM

Prostate. 2000 Jun 15;44(1):55-60. doi: 10.1002/1097-0045(20000615)44:1<55::aid-pros8>3.0.co;2-5.

Abstract

Background: The polysulfonated napthlyurea suramin has shown significant antitumor activity in patients with hormone-refractory metastatic prostate cancer. The mechanism by which suramin exerts this effect is unknown. In 1993, prostate-specific membrane antigen (PSM) was identified as a prostate biomarker that is elevated in hormone-refractory and metastatic prostate cancer. PSM is a glutamate exocarboxypeptidase capable of cleaving the terminal alpha-linked glutamate from the dipeptide N-acetyl-aspartyl-glutamate (NAAG) and the gamma-linked glutamates from folate polyglutamate.

Methods: Using a NAAG hydrolytic radioenzymatic assay, we tested whether suramin had any effect on the enzymatic activity of PSM.

Results: We demonstrate that suramin potently inhibits the enzymatic activity of PSM with a K(i) = 15 nM and 68 nM for the membrane-associated and soluble forms of PSM, respectively. In addition, we show that suramin inhibition of PSM enzyme activity displays the kinetics of a classic competitive inhibitor.

Conclusions: This is one of the most potent activities described for suramin to date and may represent a portion of its pharmacologic and/or toxicological mechanism of action.

MeSH terms

  • Antigens, Surface*
  • Antineoplastic Agents / pharmacology*
  • Carboxypeptidases / antagonists & inhibitors*
  • Carboxypeptidases / metabolism
  • Chromatography, Ion Exchange
  • Dipeptides / chemistry
  • Dose-Response Relationship, Drug
  • Glutamate Carboxypeptidase II
  • Humans
  • Male
  • Phosphates / chemistry
  • Prostatic Neoplasms / drug therapy
  • Prostatic Neoplasms / enzymology*
  • Quisqualic Acid / chemistry
  • Scintillation Counting
  • Suramin / pharmacology*
  • Tumor Cells, Cultured

Substances

  • Antigens, Surface
  • Antineoplastic Agents
  • Dipeptides
  • Phosphates
  • isospaglumic acid
  • Suramin
  • Quisqualic Acid
  • Carboxypeptidases
  • FOLH1 protein, human
  • Glutamate Carboxypeptidase II