Morphological plasticity in an infant monkey model of temporal lobe epilepsy

Epilepsia. 2000:41 Suppl 6:S70-5. doi: 10.1111/j.1528-1157.2000.tb01560.x.

Abstract

Purpose/methods: Seizures in early life are thought to contribute to the development of human temporal lobe epilepsy. To examine the consequences of early seizures, we elicited status epilepticus in immature, 5.5- to 7.0-month-old pigtailed macaques by unilateral microinfusion of bicuculline methiodide into the entorhinal cortex.

Results: This report focuses on neuropathological changes in the hippocampus. Bicuculline infusion consistently elicited limbic-like seizures with prolonged, relatively localized electrographic activity. Magnetic resonance imaging revealed enhanced signal intensity in the ipsilateral hippocampus after seizures; in some cases, there was also progressive hippocampal atrophy. Histological changes were variable; in two of five monkeys, there was significant hippocampal neuron loss, gliosis, granule cell dispersion, and mossy fiber reorganization.

Conclusions: The histopathological findings and associated magnetic resonance imaging abnormalities after bicuculline-induced status epilepticus in infant monkeys mimic common aspects of human temporal lobe epilepsy.

Publication types

  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Animals, Newborn / growth & development
  • Bicuculline / analogs & derivatives*
  • Bicuculline / pharmacology
  • Disease Models, Animal*
  • Entorhinal Cortex / drug effects
  • Entorhinal Cortex / pathology
  • Epilepsy, Temporal Lobe / pathology*
  • Hippocampus / drug effects
  • Hippocampus / pathology*
  • Macaca nemestrina
  • Magnetic Resonance Imaging
  • Mossy Fibers, Hippocampal / pathology
  • Neuronal Plasticity / physiology*
  • Status Epilepticus / chemically induced
  • Status Epilepticus / pathology
  • Temporal Lobe / pathology

Substances

  • bicuculline methiodide
  • Bicuculline