Plasma acyl-estrone levels are altered in obese women

Endocr Res. 2000 Aug;26(3):465-76. doi: 10.3109/07435800009066180.

Abstract

A group of obese women (BMI>27 kg/m2; N=73) was studied together with lean controls (BMI <27 kg/m2; N=25). Three groups were defined by the compliance with: BMI lower than 27 kg/m2, glycaemia lower than 5.5 mM and insulinaemia lower than 0.2 nM (controls, group 1, N=19). The subjects with BMI>27 kg/m2, glucose >5.5 mM and insulin >0.2 nM constituted group 3 (N=41), and those with BMI>27 with glycaemia and/or insulinaemia lower than the limits set constituted group 2 (N=32). The women in group 3 had higher fat content, BMI and fat-free mass than those in group 2 and the controls. There were no changes in most plasma parameters, such as free estrone and beta-estradiol. Leptin levels were higher in groups 2 and 3 than in controls. In controls, leptin and acyl-estrone levels were well correlated with BMI and fat content; this correlation was not found in groups 2 and 3 for acyl-estrone, although it was found for leptin. Acyl-estrone levels were lower than expected in most obese women when compared to those of controls, suggesting an altered availability or function of this hormone. In obese women, acyl-estrone levels -and probably function- are lower than expected, contrasting with maintained leptin-BMI correlations. The role of insulin in the control of body weight, perhaps through acyl estrone-mediated effects, should be re-evaluated.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Acylation
  • Adipose Tissue
  • Adult
  • Aspartate Aminotransferases / blood
  • Blood Glucose / analysis
  • Body Composition
  • Body Constitution
  • Body Mass Index
  • Estradiol / blood
  • Estrone / blood*
  • Female
  • Humans
  • Insulin / blood
  • Lactic Acid / blood
  • Leptin / analysis
  • Middle Aged
  • Obesity / blood*
  • Uric Acid / blood

Substances

  • Blood Glucose
  • Insulin
  • Leptin
  • Uric Acid
  • Estrone
  • Lactic Acid
  • Estradiol
  • Aspartate Aminotransferases