p27kip1-independent cell cycle regulation by MYC

Oncogene. 2000 Oct 5;19(42):4822-7. doi: 10.1038/sj.onc.1203879.

Abstract

MYC transcription factors are potent stimulators of cell proliferation. It has been suggested that the CDK-inhibitor p27kip1 is a critical G1 phase cell cycle target of c-MYC. We show here that mouse embryo fibroblasts deficient for both p27kip1 and the related p21cip1 are still responsive to stimulation by c-MYC and can be arrested in G1 by a dominant negative mutant of c-MYC. This growth arrest can be overruled by ectopic expression of E2F or adenovirus E1A, but not by a mutant of E1A defective for binding to retinoblastoma family proteins. We show that fibroblasts with a genetic disruption of all three retinoblastoma family members (pRb, p107 and p130) are unresponsive to a dominant negative c-MYC mutant. These data indicate that p27kip1 is not the only rate limiting cell cycle target of c-MYC and suggest that regulation of E2F is also essential for c-MYC's mitogenic activity.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenovirus E1A Proteins / genetics
  • Adenovirus E1A Proteins / physiology
  • Animals
  • Carrier Proteins*
  • Cell Cycle / physiology*
  • Cell Cycle Proteins*
  • Cyclin-Dependent Kinase Inhibitor p21
  • Cyclin-Dependent Kinase Inhibitor p27
  • Cyclins / physiology
  • DNA-Binding Proteins*
  • E2F Transcription Factors
  • Fibroblasts
  • G1 Phase / genetics
  • G1 Phase / physiology
  • Genes, Dominant
  • Genes, Retinoblastoma
  • Genes, cdc*
  • Genes, myc
  • Mice
  • Mice, Knockout
  • Microtubule-Associated Proteins / physiology*
  • Nuclear Proteins / deficiency
  • Nuclear Proteins / genetics
  • Nuclear Proteins / physiology
  • Phosphoproteins / deficiency
  • Phosphoproteins / genetics
  • Phosphoproteins / physiology
  • Proteins*
  • Proto-Oncogene Proteins c-myc / physiology*
  • Recombinant Fusion Proteins / physiology
  • Retinoblastoma Protein / deficiency
  • Retinoblastoma Protein / genetics
  • Retinoblastoma Protein / physiology
  • Retinoblastoma-Binding Protein 1
  • Retinoblastoma-Like Protein p107
  • Retinoblastoma-Like Protein p130
  • S Phase / genetics
  • S Phase / physiology
  • Transcription Factor DP1
  • Transcription Factors / genetics
  • Transcription Factors / physiology
  • Tumor Suppressor Proteins*

Substances

  • Adenovirus E1A Proteins
  • Arid4a protein, mouse
  • Carrier Proteins
  • Cdkn1a protein, mouse
  • Cdkn1b protein, mouse
  • Cell Cycle Proteins
  • Cyclin-Dependent Kinase Inhibitor p21
  • Cyclins
  • DNA-Binding Proteins
  • E2F Transcription Factors
  • Microtubule-Associated Proteins
  • Nuclear Proteins
  • Phosphoproteins
  • Proteins
  • Proto-Oncogene Proteins c-myc
  • Rbl1 protein, mouse
  • Rbl2 protein, mouse
  • Recombinant Fusion Proteins
  • Retinoblastoma Protein
  • Retinoblastoma-Binding Protein 1
  • Retinoblastoma-Like Protein p107
  • Retinoblastoma-Like Protein p130
  • Transcription Factor DP1
  • Transcription Factors
  • Tumor Suppressor Proteins
  • Cyclin-Dependent Kinase Inhibitor p27