Thyroid hormone receptor beta-deficient mice show complete loss of the normal cholesterol 7alpha-hydroxylase (CYP7A) response to thyroid hormone but display enhanced resistance to dietary cholesterol

Mol Endocrinol. 2000 Nov;14(11):1739-49. doi: 10.1210/mend.14.11.0548.

Abstract

Thyroid hormone (T3) influences hepatic cholesterol metabolism, and previous studies have established an important role of this hormone in the regulation of cholesterol 7alpha-hydroxylase (CYP7A), the rate-limiting enzyme in the synthesis of bile acids. To evaluate the respective contribution of thyroid hormone receptors (TR) alpha1 and beta in this regulation, the responses to 2% dietary cholesterol and T3 were studied in TRalpha1 and TRbeta knockout mice under hypo- and hyperthyroid conditions. Our experiments show that the normal stimulation in CYP7A activity and mRNA level by T3 is lost in TRbeta-/- but not in TRalpha1-/-mice, identifying TRbeta as the mediator of T3 action on CYP7A and, consequently, as a major regulator of cholesterol metabolism in vivo. Somewhat unexpectedly, T3-deficient TRbeta-/- mice showed an augmented CYP7A response after challenge with dietary cholesterol, and these animals did not develop hypercholesterolemia to the extent as did wild-type (wt) controls. The latter results lend strong support to the concept that TRs may exert regulatory effects in vivo independent of T3.

Publication types

  • Comparative Study
  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.

MeSH terms

  • Animals
  • Cholesterol / blood
  • Cholesterol / metabolism
  • Cholesterol 7-alpha-Hydroxylase / drug effects
  • Cholesterol 7-alpha-Hydroxylase / metabolism*
  • Cholesterol, Dietary / pharmacology*
  • DNA-Binding Proteins
  • Hydroxymethylglutaryl CoA Reductases / genetics
  • Hydroxymethylglutaryl CoA Reductases / metabolism
  • Hypercholesterolemia / chemically induced
  • Hypercholesterolemia / genetics
  • Iodide Peroxidase / genetics
  • Iodide Peroxidase / metabolism
  • Lipoproteins, HDL / blood
  • Lipoproteins, LDL / blood
  • Liver / metabolism
  • Liver X Receptors
  • Male
  • Mice
  • Mice, Knockout
  • Orphan Nuclear Receptors
  • Receptors, Cytoplasmic and Nuclear / genetics
  • Receptors, Cytoplasmic and Nuclear / metabolism
  • Receptors, Thyroid Hormone / genetics*
  • Thyroid Hormones / metabolism
  • Thyroid Hormones / pharmacology*
  • Triiodothyronine / metabolism
  • Triiodothyronine / pharmacology

Substances

  • Cholesterol, Dietary
  • DNA-Binding Proteins
  • Lipoproteins, HDL
  • Lipoproteins, LDL
  • Liver X Receptors
  • Orphan Nuclear Receptors
  • Receptors, Cytoplasmic and Nuclear
  • Receptors, Thyroid Hormone
  • Thyroid Hormones
  • Triiodothyronine
  • Cholesterol
  • Hydroxymethylglutaryl CoA Reductases
  • iodothyronine deiodinase type I
  • Iodide Peroxidase
  • Cholesterol 7-alpha-Hydroxylase