Positive and negative regulation of IL-12 receptor expression of naive CD4(+) T cells by CD28/CD152 co-stimulation

Eur J Immunol. 2000 Nov;30(11):3171-80. doi: 10.1002/1521-4141(200011)30:11<3171::AID-IMMU3171>3.0.CO;2-C.

Abstract

IL-12 is a critical cytokine for polarizing naive CD4(+) T cells toward Th1 subset. Therefore, it is important to elucidate the mechanism of IL-12R expression of naive CD4(+) T cells. In this report, we present evidence to show that expression of both IL-12Rbeta1 and beta2 mRNA is regulated by signals mediated through CD28 and CD152. Naive CD4(+) T cells stimulated with anti-CD3 alone neither expressed IL-12Rbeta2 mRNA nor bound detectable level of rIL-12, although they expressed a very low level of IL-12Rbeta1 mRNA when stimulated with a high dose of anti-CD3. Expression of IL-12Rbeta1 and beta2 mRNA was induced by the co-ligation of CD3 and CD28, and it was down-regulated by the ligation of CD152. CD28 ligation induced not only IL-12Rbeta1 and beta2 mRNA expression, but also enhanced IFN-gammaR to mediate up-regulation of IL-12R by IFN-gamma.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Abatacept
  • Animals
  • Antigens, CD
  • Antigens, Differentiation / immunology*
  • CD28 Antigens / immunology*
  • CD4-Positive T-Lymphocytes / immunology*
  • CHO Cells
  • CTLA-4 Antigen
  • Cricetinae
  • Gene Expression Regulation / immunology
  • Immunoconjugates*
  • Ligands
  • Mice
  • Mice, Inbred C57BL
  • Receptors, Interleukin / biosynthesis
  • Receptors, Interleukin / immunology*
  • Receptors, Interleukin-12
  • Signal Transduction / immunology

Substances

  • Antigens, CD
  • Antigens, Differentiation
  • CD28 Antigens
  • CTLA-4 Antigen
  • Ctla4 protein, mouse
  • Il12rb2 protein, mouse
  • Immunoconjugates
  • Ligands
  • Receptors, Interleukin
  • Receptors, Interleukin-12
  • Abatacept