Collagen IV deposits do not prevent regrowing axons from penetrating the lesion site in spinal cord injury

J Neurosci Res. 2000 Dec 1;62(5):686-91. doi: 10.1002/1097-4547(20001201)62:5<686::AID-JNR7>3.0.CO;2-L.

Abstract

Scarring is suggested to impede axon regrowth across the lesion site in the injured adult mammalian central nervous system. Collagen Type IV, as a major component of the scar formed after injury, is an impediment for successful axonal regeneration and a decrease in its amount is a prerequisite for regrowing axons to cross the lesion in the postcommissural fornix in the injured adult rat (Stichel et al. [1999] Neurosci. 93:321-333). The aim of the present study was to analyze the relationship between collagen IV deposits and regrowing axons at various times after dorsal hemi-section of the adult rat spinal cord. Immunohistochemical double staining revealed that penetrating neurofilament-positive axons and collagen IV deposits were co-localized in the lesion site in the initial stages of axonal sprouting (between 7 and 14 days post-operatively) and were still present 1 and 2 months post-operatively. Interestingly, collagen IV-immunoreactive areas located around cystic cavities formed at the site of injury 1 month post-operatively, were devoid of axons. In conclusion, our observations indicate that collagen IV deposits after spinal cord injury do not prevent neurofilament-positive regrowing axons from penetrating the lesion site.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Axons* / metabolism
  • Collagen / metabolism*
  • Male
  • Microscopy, Fluorescence
  • Nerve Regeneration*
  • Rats
  • Rats, Wistar
  • Spinal Cord / metabolism
  • Spinal Cord / physiopathology
  • Spinal Cord Injuries / metabolism*
  • Spinal Cord Injuries / physiopathology

Substances

  • Collagen