Opening of the active site of Clostridium perfringens alpha-toxin may be triggered by membrane binding

Int J Med Microbiol. 2000 Oct;290(4-5):357-61. doi: 10.1016/s1438-4221(00)80040-5.

Abstract

On the basis of amino acid sequence homologies with other phospholipases C, the alpha-toxin of Clostridium perfringens was predicted to be a two-domain protein. Using truncated forms of alpha-toxin the phospholipase C active site was shown to be located in the amino-terminal domain. Crystallographic studies have confirmed this organisation and have also revealed that the carboxy-terminal domain is structurally similar to the phospholipid-binding domains in eukaryotic proteins. This information has been used to devise a model predicting how alpha-toxin interacts with membranes via calcium-mediated recognition of phospholipid head groups and the interaction of hydrophobic amino acids with the phospholipid tail group. The binding of alpha-toxin to membranes appears to result in the opening of the active site allowing hydrolysis of membrane phospholipids.

Publication types

  • Review

MeSH terms

  • Animals
  • Bacterial Toxins / chemistry*
  • Bacterial Toxins / metabolism
  • Bacterial Toxins / toxicity
  • Binding Sites
  • Calcium-Binding Proteins*
  • Cell Membrane / metabolism
  • Clostridium perfringens / pathogenicity*
  • Crystallization
  • Humans
  • Protein Conformation
  • Type C Phospholipases / chemistry*
  • Type C Phospholipases / metabolism
  • Type C Phospholipases / toxicity

Substances

  • Bacterial Toxins
  • Calcium-Binding Proteins
  • Type C Phospholipases
  • alpha toxin, Clostridium perfringens