Dissociation of LPS-induced monocytic ex vivo production of granulocyte colony-stimulating factor (G-CSF) and TNF-alpha in patients with septic shock

Cytokine. 2001 Jan 7;13(1):51-54. doi: 10.1006/cyto.2000.0796.

Abstract

Over a 6 month period, in 192 patients admitted to the intensive care unit (ICU), a longitudinal analysis of whole blood lipopolysaccharide (LPS)-induced ex vivo cytokine production was performed on a daily basis until discharge from the ICU or death. Twenty-one patients with proven infections were in septic shock for the first time and for at least 3 days' duration. Ex vivo LPS-inducible release of granulocyte colony-stimulating factor (G-CSF) was upregulated and that of TNF-alpha was downregulated in patients with septic shock, regardless whether they survived or died. In conclusion, LPS-induced ex vivo TNF-alpha and G-CSF cytokine release by monocytes is regulated differentially in patients with septic shock. Since upregulation of LPS-induced production of G-CSF occurred earlier in survivors than in non-survivors, rapidly elevated and sustained G-CSF responsiveness may contribute to survival in septic shock.

MeSH terms

  • Adult
  • Aged
  • Aged, 80 and over
  • Critical Care
  • Down-Regulation / drug effects
  • Female
  • Granulocyte Colony-Stimulating Factor / blood*
  • Humans
  • In Vitro Techniques
  • Lipopolysaccharides / pharmacology*
  • Male
  • Middle Aged
  • Monocytes / drug effects*
  • Monocytes / metabolism*
  • Prognosis
  • Shock, Septic / blood*
  • Shock, Septic / mortality
  • Tumor Necrosis Factor-alpha / metabolism*
  • Up-Regulation / drug effects

Substances

  • Lipopolysaccharides
  • Tumor Necrosis Factor-alpha
  • Granulocyte Colony-Stimulating Factor