Functional evidence for an ovarian cancer tumor suppressor gene on chromosome 22 by microcell-mediated chromosome transfer

Oncogene. 2000 Dec 14;19(54):6277-85. doi: 10.1038/sj.onc.1204013.

Abstract

The identity of many tumor suppressor genes important in epithelial ovarian cancer tumorigenesis remains unknown. In an effort to localize a novel tumor suppressor on chromosome 22, a psv2neo tagged human chromosome 22 was transferred into the malignant epithelial ovarian cancer cell line, SKOv-3, by microcell-mediated chromosome transfer. Complete suppression of the transformed phenotype was observed in 16 of 18 individual microcell hybrid clones as evidenced by the complete abrogation of cell growth under anchorage-independent conditions. In vitro doubling times were also dramatically reduced, as was the ability to form subcutaneous tumors in CD1 nu/nu mice. Only one polymorphic marker, D22S429, segregated with decreased transformation and tumorigenic potential, suggesting that an unrecognized tumor suppressor may localize to chromosome 22q11-q12. These data provide functional support for the presence of a novel tumor suppressor locus (or loci) on chromosome 22 that is important in ovarian cancer tumorigenesis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Carcinoma / genetics*
  • Carcinoma / pathology
  • Cell Division
  • Cell Fusion
  • Chromosomes, Human, Pair 22*
  • Female
  • Gene Transfer Techniques*
  • Genes, Tumor Suppressor*
  • Genetic Markers
  • Humans
  • Hybrid Cells
  • Mice
  • Mice, Nude
  • Microsatellite Repeats
  • Ovarian Neoplasms / genetics*
  • Ovarian Neoplasms / pathology
  • Phenotype
  • Suppression, Genetic
  • Tumor Cells, Cultured

Substances

  • Genetic Markers