Isolation and characterization of simian immunodeficiency virus variants that are resistant to nonnucleoside reverse transcriptase inhibitors

Arch Virol. 2000;145(12):2481-92. doi: 10.1007/s007050070003.

Abstract

Non-nucleoside reverse transcriptase inhibitors (NNRTIs) act quite specifically on human immunodeficiency virus type 1 (HIV-1). In general, they are not effective on human immunodeficiency virus type 2 (HIV-2) or simian immunodeficiency virus (SIV). Only SIV strains from African green monkeys are sensitive to several NNRTIs. Here we isolated NNRTI- and 3TC-resistant SIVagm variants. Viruses resistant to delavirdine contained V112I and M231I substitutions, while those resistant to 3TC contained a M 185I substitution. These amino acids are highly conserved in HIV-1, HIV-2, SIVmac and SIVagm, and the M184I (M185I in SIVagm) substitution was observed in 3TC-resistant HIV-1 and SIVmac. The roles of the observed mutations in NNRTI-resistance of SIVagm and HIV-1 were further confirmed by site-directed mutagenesis. The present results have provided a new insight into the common mechanism of sensitivity of HIV- 1 and SIVagm to NNRTIs.

Publication types

  • Comparative Study

MeSH terms

  • Amino Acid Sequence
  • Amino Acid Substitution
  • Delavirdine / pharmacology
  • Drug Resistance, Microbial
  • HeLa Cells
  • Humans
  • Lamivudine / pharmacology
  • Molecular Sequence Data
  • Point Mutation
  • Reverse Transcriptase Inhibitors / pharmacology*
  • Sequence Alignment
  • Simian Immunodeficiency Virus / drug effects*
  • Simian Immunodeficiency Virus / genetics
  • Simian Immunodeficiency Virus / isolation & purification
  • Structure-Activity Relationship

Substances

  • Reverse Transcriptase Inhibitors
  • Lamivudine
  • Delavirdine