Hyperpolarizing shift by quinine in the steady-state inactivation curve of delayed rectifier-type potassium current in bullfrog sympathetic neurons

Neurosci Lett. 2001 Mar 9;300(2):87-90. doi: 10.1016/s0304-3940(01)01554-3.

Abstract

Whole-cell recordings were made from dissociated bullfrog sympathetic neurons to examine the actions of quinine (1-100 microM) on the steady-state activation and inactivation curves of a delayed rectifier-type potassium current (I(K)). Quinine (EC50 approximately 8 microM) caused a hyperpolarizing shift (approximately 31 mV with 30 microM) in the inactivation curve of I(K) without significantly affecting its activation curve. Quinine (20 microM) was without effects on the voltage-dependence of a rapidly-inactivating A-type potassium current (I(A)). It is concluded that quinine can selectively modulate the voltage-dependence of I(K) in amphibian autonomic neurons.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Cell Membrane / drug effects*
  • Cell Membrane / metabolism
  • Dose-Response Relationship, Drug
  • Ganglia, Sympathetic / cytology
  • Ganglia, Sympathetic / drug effects*
  • Ganglia, Sympathetic / metabolism
  • Kinetics
  • Membrane Potentials / drug effects*
  • Membrane Potentials / physiology
  • Muscle Relaxants, Central / pharmacology*
  • Neurons / drug effects*
  • Neurons / metabolism
  • Potassium Channels / drug effects*
  • Potassium Channels / metabolism
  • Quinine / pharmacology*
  • Rana catesbeiana

Substances

  • Muscle Relaxants, Central
  • Potassium Channels
  • Quinine