Possible cellular mechanism accounting for the cell type-specific vulnerability in Huntington's disease

Funct Neurol. 2000 Oct-Dec;15(4):253-8.

Authors

D Centonze¹, P Giacomini, M Tolu, G Bernardi, P Calabresi

Affiliation

¹ Neurological Clinic, Department of Neurosciences, University Tor Vergata and IRCCS S. Lucia Hospital, Rome, Italy.

PMID: 11213528

No abstract available

Publication types

Research Support, Non-U.S. Gov't

MeSH terms

Calcium Channels / metabolism
Cholinergic Fibers / metabolism
Cholinergic Fibers / pathology
Corpus Striatum / metabolism
Corpus Striatum / pathology
Glucose / deficiency
Humans
Huntington Disease / metabolism*
Huntington Disease / pathology*
Intracellular Membranes / metabolism
Ion Transport / physiology
Nerve Degeneration / metabolism*
Nerve Degeneration / pathology*
Oxygen / metabolism
Receptors, Glutamate / metabolism
Receptors, N-Methyl-D-Aspartate / metabolism

Substances

Calcium Channels
Receptors, Glutamate
Receptors, N-Methyl-D-Aspartate
Glucose
Oxygen