Transgenic models of ovarian failure

J Soc Gynecol Investig. 2001 Jan-Feb;8(1 Suppl Proceedings):S30-3. doi: 10.1016/s1071-5576(00)00103-9.

Abstract

Reproductive development and function are a complex process requiring the coordinate interactions of multiple extragonadal and intragonadal factors. Peptide hormones from the hypothalamus (eg, gonadotropin releasing hormone) and the pituitary (eg, follicle stimulating hormone, luteinizing hormone, oxytocin) as well as growth factors and steroids from the gonads (eg, inhibins, growth differentiation factor-9, progesterone, estradiol, testosterone) play key functions in regulating specific aspects of reproduction. Our laboratory used spontaneous mutant mice and transgenic mouse models to address the essential roles of some of these factors in the propagation of the mammalian species. This review summarizes some of our initial studies to understand this process.

Publication types

  • Research Support, Non-U.S. Gov't
  • Research Support, U.S. Gov't, P.H.S.
  • Review

MeSH terms

  • Animals
  • Bone Morphogenetic Protein 15
  • Female
  • Growth Differentiation Factor 9
  • Growth Substances / genetics
  • Growth Substances / physiology
  • Inhibins / genetics
  • Inhibins / physiology
  • Intercellular Signaling Peptides and Proteins*
  • Male
  • Mice
  • Mice, Knockout
  • Mice, Transgenic
  • Mutation
  • Oxytocin / genetics
  • Oxytocin / physiology
  • Primary Ovarian Insufficiency / genetics*
  • Superoxide Dismutase / genetics
  • Superoxide Dismutase / physiology

Substances

  • Bmp15 protein, mouse
  • Bone Morphogenetic Protein 15
  • Gdf9 protein, mouse
  • Growth Differentiation Factor 9
  • Growth Substances
  • Intercellular Signaling Peptides and Proteins
  • Oxytocin
  • Inhibins
  • Superoxide Dismutase