Spontaneous retinopathy in HLA-A29 transgenic mice

Proc Natl Acad Sci U S A. 2001 Feb 27;98(5):2572-6. doi: 10.1073/pnas.051595998. Epub 2001 Feb 13.

Abstract

Humans who have inherited the class I major histocompatibility allele HLA-A29 have a markedly increased relative risk of developing the eye disease termed birdshot chorioretinopathy. This disease affecting adults is characterized by symmetrically scattered, small, cream-colored spots in the fundus associated with retinal vasculopathy and inflammatory signs causing damage to the ocular structures, leading regularly to visual loss. To investigate the role of HLA-A29 in this disease, we introduced the HLA-A29 gene into mice. Aging HLA-A29 transgenic mice spontaneously developed retinopathy, showing a striking resemblance to the HLA-A29-associated chorioretinopathy. These results strongly suggest that HLA-A29 is involved in the pathogenesis of this disease. Elucidation of the role of HLA-A29 should be assisted by this transgenic model.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Animals
  • Flow Cytometry
  • HLA-A Antigens / genetics
  • HLA-A Antigens / physiology*
  • Mice
  • Mice, Inbred BALB C
  • Mice, Transgenic
  • Retinal Diseases / immunology*
  • Retinal Diseases / pathology

Substances

  • HLA-A Antigens
  • HLA-A29 antigen