Ribosomal protein S5 interacts with the internal ribosomal entry site of hepatitis C virus

J Biol Chem. 2001 Jun 15;276(24):20824-6. doi: 10.1074/jbc.C100206200. Epub 2001 Apr 30.

Abstract

Translational initiation of hepatitis C virus (HCV) genome RNA occurs via its highly structured 5' noncoding region called the internal ribosome entry site (IRES). Recent studies indicate that HCV IRES and 40 S ribosomal subunit form a stable binary complex that is believed to be important for the subsequent assembly of the 48 S initiation complex. Ribosomal protein (rp) S9 has been suggested as the prime candidate protein for binding of the HCV IRES to the 40 S subunit. RpS9 has a molecular mass of approximately 25 kDa in UV cross-linking experiments. In the present study, we examined the approximately 25-kDa proteins of the 40 S ribosome that form complexes with the HCV IRES upon UV cross-linking. Immunoprecipitation with specific antibodies against two 25-kDa 40 S proteins, rpS5 and rpS9, clearly identified rpS5 as the protein bound to the IRES. Thus, our results support rpS5 as the critical element in positioning the HCV RNA on the 40 S ribosomal subunit during translation initiation.

MeSH terms

  • 5' Untranslated Regions / genetics*
  • Amino Acid Sequence
  • Binding Sites
  • Hepacivirus / genetics
  • Hepacivirus / physiology*
  • Molecular Sequence Data
  • Molecular Weight
  • Peptide Chain Initiation, Translational
  • RNA, Viral / genetics*
  • Ribosomal Protein S9
  • Ribosomal Proteins / chemistry
  • Ribosomal Proteins / metabolism*
  • Ribosomes / metabolism*
  • Ribosomes / virology*

Substances

  • 5' Untranslated Regions
  • RNA, Viral
  • Ribosomal Protein S9
  • Ribosomal Proteins
  • ribosomal protein S40
  • ribosomal protein S5