Inflammatory stimuli increase prostaglandin D synthase levels in cerebrospinal fluid of rats

Neuroreport. 2001 May 8;12(6):1161-5. doi: 10.1097/00001756-200105080-00022.

Abstract

To examine the possibility that prostaglandin D synthase in CSF is involved in the brain responses to inflammation, we monitored the enzyme levels before and after administration of a bacterial endotoxin lipopolysaccharide (LPS) or proinflammatory cytokines. After an i.p. injection of LPS, the synthase levels began to rise within 1 day, peaked at 2 days and returned to the basal by 5 days after injection. The enzyme levels increased in a similar manner in response to tumor necrosis factor-alpha or interleukin (IL)-1 beta injected i.v. Centrally administered IL-1 beta had a similar effect. These findings support the hypothesis that PGD synthase in CSF participates in the brain responses to inflammation.

MeSH terms

  • Animals
  • Inflammation / chemically induced*
  • Inflammation / enzymology*
  • Injections, Intraperitoneal
  • Injections, Intravenous
  • Injections, Intraventricular
  • Interleukin-1 / administration & dosage
  • Intramolecular Oxidoreductases / cerebrospinal fluid*
  • Lipocalins
  • Lipopolysaccharides / administration & dosage
  • Male
  • Rats
  • Rats, Wistar
  • Tumor Necrosis Factor-alpha / administration & dosage

Substances

  • Interleukin-1
  • Lipocalins
  • Lipopolysaccharides
  • Tumor Necrosis Factor-alpha
  • Intramolecular Oxidoreductases
  • prostaglandin R2 D-isomerase