Possible role of uncoupling protein in regulation of myocardial energy metabolism in aortic regurgitation model rats

FASEB J. 2001 May;15(7):1206-8. doi: 10.1096/fj.000569fje.
No abstract available

MeSH terms

  • Adenosine Triphosphate / metabolism*
  • Animals
  • Aortic Valve Insufficiency / metabolism*
  • Atrial Natriuretic Factor / genetics
  • Atrial Natriuretic Factor / metabolism
  • Blotting, Northern
  • Cardiac Output, Low / physiopathology
  • Disease Models, Animal
  • Humans
  • Ion Channels
  • Male
  • Membrane Transport Proteins*
  • Mitochondria, Heart / metabolism
  • Mitochondrial Proteins*
  • Muscle, Skeletal / physiology
  • Myocardium / metabolism*
  • Phosphocreatine / metabolism
  • Proteins / genetics
  • Proteins / metabolism*
  • RNA, Messenger / metabolism
  • Rats
  • Rats, Sprague-Dawley
  • Tumor Necrosis Factor-alpha / genetics
  • Tumor Necrosis Factor-alpha / metabolism
  • Uncoupling Agents / metabolism*
  • Uncoupling Protein 2

Substances

  • Ion Channels
  • Membrane Transport Proteins
  • Mitochondrial Proteins
  • Proteins
  • RNA, Messenger
  • Tumor Necrosis Factor-alpha
  • Uncoupling Agents
  • Uncoupling Protein 2
  • Phosphocreatine
  • Atrial Natriuretic Factor
  • Adenosine Triphosphate