Induction of VEGF in perivascular cells defines a potential paracrine mechanism for endothelial cell survival

FASEB J. 2001 May;15(7):1239-41. doi: 10.1096/fj.00-0693fje.
No abstract available

MeSH terms

  • Animals
  • Anticoagulants / pharmacology
  • Apoptosis / physiology
  • Becaplermin
  • Cell Survival
  • Cells, Cultured
  • Culture Media, Conditioned
  • Endothelial Growth Factors / biosynthesis*
  • Endothelial Growth Factors / genetics
  • Endothelial Growth Factors / metabolism
  • Endothelium, Vascular / cytology
  • Endothelium, Vascular / drug effects
  • Endothelium, Vascular / metabolism*
  • Gene Expression Regulation*
  • Humans
  • Indoles / pharmacology
  • Lymphokines / biosynthesis*
  • Lymphokines / genetics
  • Lymphokines / metabolism
  • Mice
  • Models, Biological
  • Muscle, Smooth, Vascular / cytology
  • Muscle, Smooth, Vascular / drug effects
  • Muscle, Smooth, Vascular / metabolism*
  • Oxindoles
  • Paracrine Communication*
  • Platelet-Derived Growth Factor / pharmacology*
  • Propionates
  • Proto-Oncogene Proteins c-sis
  • Pyrroles / pharmacology
  • RNA, Messenger / metabolism
  • Tumor Cells, Cultured
  • Vascular Endothelial Growth Factor A
  • Vascular Endothelial Growth Factors

Substances

  • Anticoagulants
  • Culture Media, Conditioned
  • Endothelial Growth Factors
  • Indoles
  • Lymphokines
  • Oxindoles
  • Platelet-Derived Growth Factor
  • Propionates
  • Proto-Oncogene Proteins c-sis
  • Pyrroles
  • RNA, Messenger
  • Vascular Endothelial Growth Factor A
  • Vascular Endothelial Growth Factors
  • Becaplermin
  • orantinib