[Study on tumor necrosis factor and pathogenesis of pregnancy induced hypertension]

Zhonghua Fu Chan Ke Za Zhi. 1999 Jun;34(6):339-41.
[Article in Chinese]

Abstract

Objective: To study the role of tumor necrosis factor (TNF-alpha) in the pathogenesis of pregnancy induced hypertension (PIH).

Methods: Radioimmunoassay was used to measure the levels of TNF-alpha in 25 severe PIH patients and 25 normal pregnant women. Umbilical vein endothelial cells were cultured with TNF-alpha (500 U/ml) or without TNF-alpha. The concentration of endothelin-1 (ET-1), 6-ket-PGF1 alpha, nitrite (NO2-), expression of fibronectin(FN) and white blood cells adhesion to the surface of endothelial cells test were measured.

Results: The levels of TNF-alpha in serum of severe PIH patients were significantly higher than that of normal pregnant women (P < 0.05). In endothelium culture supernatant with TNF-alpha group, synthesis of ET-1, NO2- and 6-ket-PGF1 alpha increased, expression of FN on the surface of endothelial cells decreased, white blood cells adhesion to endothelial cells increased. There was significant difference between TNF-alpha group and control group.

Conclusion: TNF-alpha may be involved in the pathogenesis of PIH.

Publication types

  • English Abstract
  • Research Support, Non-U.S. Gov't

MeSH terms

  • 6-Ketoprostaglandin F1 alpha / metabolism
  • Adult
  • Cells, Cultured
  • Endothelin-1 / metabolism
  • Endothelium, Vascular / cytology
  • Endothelium, Vascular / metabolism
  • Female
  • Humans
  • Infant, Newborn
  • Pre-Eclampsia / blood
  • Pre-Eclampsia / etiology*
  • Pregnancy
  • Tumor Necrosis Factor-alpha / metabolism*
  • Tumor Necrosis Factor-alpha / pharmacology
  • Umbilical Veins / cytology*

Substances

  • Endothelin-1
  • Tumor Necrosis Factor-alpha
  • 6-Ketoprostaglandin F1 alpha