Cyclin E mRNA overexpression in epithelial ovarian cancers: inverse correlation with p53 protein accumulation

J Soc Gynecol Investig. 2001 May-Jun;8(3):179-85.

Abstract

Objective: We investigated the relationship between cyclin E mRNA overexpression and p53 protein accumulation in epithelial ovarian cancers.

Methods: mRNA was isolated and cDNA was prepared from 36 epithelial ovarian tumors (three adenomas, three low malignant potential tumors, and 30 carcinomas), and six normal ovaries. The cyclin E mRNA expression levels relative to an internal control, beta-tubulin, were determined by semiquantitative polymerase chain reaction (PCR). Cyclin E and p53 protein expression in ovarian cancer tissues were examined by immunohistochemistry using the same series of samples. Fisher exact test of significance and an unpaired t test were used for statistical analysis.

Results: Considerable levels of cyclin E mRNA were detected in all normal ovaries and ovarian tumor samples examined by semiquantitative PCR amplification. mRNA levels of cyclin E were significantly higher in nine of 30 (30%) ovarian cancers compared with those in normal ovaries. The immunohistochemical expression of cyclin E protein was confirmed in the nuclei of tumor cells in 13 of 30 (43%) ovarian cancers. p53 protein accumulation was detected in 12 of 30 (40%) ovarian cancers examined. There was a significant inverse correlation between cyclin E mRNA overexpression and p53 protein accumulation (P <.01, Fisher exact test).

Conclusions: Cyclin E mRNA overexpression frequently occurs in ovarian cancers without p53 protein accumulation. Cyclin E might have an important effect on the development of a limited number of ovarian cancers.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Adenoma / chemistry
  • Adenoma / metabolism*
  • Carcinoma / metabolism*
  • Cyclin E / analysis
  • Cyclin E / genetics*
  • Female
  • Gene Expression*
  • Humans
  • Immunohistochemistry
  • Ovarian Neoplasms / chemistry
  • Ovarian Neoplasms / metabolism*
  • Ovary / chemistry
  • Polymerase Chain Reaction
  • RNA, Messenger / analysis
  • Tubulin / genetics
  • Tumor Suppressor Protein p53 / chemistry
  • Tumor Suppressor Protein p53 / metabolism*

Substances

  • Cyclin E
  • RNA, Messenger
  • Tubulin
  • Tumor Suppressor Protein p53