Expression of a newly defined tumor-rejection antigen SART3 in musculoskeletal tumors and induction of HLA class I-restricted cytotoxic T lymphocytes by SART3-derived peptides

J Orthop Res. 2001 May;19(3):346-51. doi: 10.1016/S0736-0266(00)90031-7.

Abstract

We recently reported that a SART3 tumor-rejection antigen possessing tumor epitopes is capable of inducing HLA class 1-restricted and tumor-specific cytotoxic T lymphocytes in cancer patients. We studied the expression of the SART3 protein in musculoskeletal tumors to find a molecule for potential use in tumor-specific immunotherapy. The SART3 was detected at protein levels in 100% of the osteosarcoma cell lines (n = 20), in 50% of the musculoskeletal tumor tissue specimens (n = 32), and at notable levels in 67% of osteosarcoma tissues (n = 9) and malignant fibrous histiocytosis tissues (n = 9), respectively. SART3-derived peptides at positions 109-118 and 315-323 induced HLA-A24-restricted tumor-specific cytoxic T lymphocytes from peripheral blood mononuclear cells of patients with osteosarcoma or malignant fibrous histiocytosis. These peptide-induced cytotoxic T lymphocytes recognized HLA-A24+ SART3+ osteosarcoma cells but not HLA-A24 or SART3 cells. These results suggest that the SART3 protein and its derived peptides could be molecules appropriate for use in specific immunotherapies for approximately 60% of HLA-A24+ patients with osteosarcoma or malignant fibrous histiocytosis.

Publication types

  • Research Support, Non-U.S. Gov't

MeSH terms

  • Antigens, Neoplasm / biosynthesis*
  • Bone Neoplasms / metabolism*
  • Bone Neoplasms / pathology
  • Cell Survival / drug effects
  • Cytotoxicity Tests, Immunologic
  • Histiocytoma, Benign Fibrous / metabolism*
  • Histiocytoma, Benign Fibrous / pathology
  • Histocompatibility Antigens Class I / immunology
  • Humans
  • Immunotherapy
  • Osteosarcoma / metabolism*
  • Osteosarcoma / pathology
  • Peptide Fragments / pharmacology*
  • RNA-Binding Proteins / biosynthesis*
  • T-Lymphocytes, Cytotoxic / drug effects*
  • T-Lymphocytes, Cytotoxic / immunology
  • Tumor Cells, Cultured

Substances

  • Antigens, Neoplasm
  • Histocompatibility Antigens Class I
  • Peptide Fragments
  • RNA-Binding Proteins
  • SART3 protein, human